Treatment combo Sessions of Modern Medicine in Digital format

Treatment combo Sessions of Modern Medicine in Digital format - A

The frequencies used in these sessions are based upon Rife sets for resonant therapy devices masked in Algorithmic piano music.

More information regarding the items in the list is given below the list.

List of Treatment combo Sessions of Modern Medicine in Digital format for problems/products available at us for just Rs. 1,000/- for any 5 sessions from any one or multiple Treatment combo Sessions for 30 doses per session (2 times a day for 15 days) in max 15 days.

1) Acidosis
2) Acne
3) Acromegaly
4) Acute Inflammatory Neuropathy
5) Addictions Alcohol-General
6) Addictions Drug-General
7) Addison Disease
8) Adenoma
9) Adenomatous Polyposis Coli
10) Adenomyosis
11) Adenovirus Infections
12) Adhesions Pelvic
13) Adiposis Dolorosa
14) Adnexitis
15) Adrenal Gland Diseases
16) Adrenal Hyperplasia Congenital
17) Adrenoleukodystrophy
18) African Sleeping Sickness
19) Agammaglobulinemia
20) Aganglionosis Colonic
21) Aicardi Syndrome
22) AIDS HIV
23) Aldosteronism
24) Alexander Disease
25) Alkalosis
26) Alkaptonuria
27) All Diabetes-Comprehensive
28) All Microbes And Disease
29) Allergic Granulomatous Angiitis
30) Alopecia
31) Alpha-Antitrypsin Deficiency
32) Altitude Sickness
33) Amblyopia
34) Amino Acid Metabolism
35) Amniotic Band Syndrome
36) Amyloidosis
37) Amyotrophic Lateral Sclerosis
38) Analgesia
39) Anemia
40) Anemia Fanconi
41) Anemia Megaloblastic
42) Anemia Pernicious
43) Aneurysm
44) Angina Pectoris
45) Angiofibroma
46) Angiokeratoma Corporis Diffusum
47) Angiolymphoid
48) Angioma
49) Anhydrosis
50) Aniridia
51) Anisakiasis
52) Anisocoria
53) Anophthalmos
54) Antiphospholipid Syndrome
55) Antiseptic Effect
56) Antithrombin-III Deficiency
57) Anus Diseases
58) Aortic Valve Stenosis
59) Aortitis Syndrome
60) Aplastic Anemia
61) Apnea
62) Appendicitis
63) Aprosencephaly
64) Arachnoid Cysts
65) Arachnoiditis
66) Arbovirus Infections
67) Arm Injuries
68) Arnold-Chiari Malformation
69) Arrhythmogenic Cardiomyopathy
70) Arteriosclerosis
71) Arteriovenous Malformations
72) Arteritis Temporal
73) Arthritis
74) Arthritis Juvenile Chronic
75) Arthritis Reactive
76) Arthritis Rheumatoid
77) Arthrogryposis
78) Asbestosis
79) Ascariasis
80) Ascorbic Acid Deficiency
81) Asphyxia
82) Asthma
83) Asthma Plus Allergies-Comprehensive
84) Astigmatism
85) Astrocytoma
86) Ataxia Telangiectasia
87) Atrial Fibrillation
88) Autonomic Nervous System Diseases
89) Avitaminosis

* Acidosis, increased acidity in the blood and other body tissues.

* Acne, often found in people who eat less fruit than the average. A supplement of 500mg/day of vitamin B5 for a month or two will solve most types of acnes. Follow-up getting into the habit of eating more fruit.

* Acromegaly is a rare condition in which the body makes too much growth hormone. This causes various symptoms which slowly develop over several years. The most noticeable symptoms are that the hands and feet become larger and features of the face may become more prominent. The cause is usually a small non-cancerous growth (tumour) in the pituitary gland. Rarely, acromegaly is caused by an excess of GHRH which is made in the hypothalamus. This stimulates the cells in the pituitary gland to make too much growth hormone. Very rarely, other tumours in the body can make growth hormone. Common symptoms of acromegaly are: enlarged bones in the face, feet, and hands, excessive hair growth in women, an enlarged jaw or tongue, a prominent brow, excessive growth spurts, which are more common in people who've had abnormal growth before adolescence, weight gain, swollen and painful joints that limit movement, spaces between the teeth, splayed fingers and toes, a hoarse, deep voice, fatigue, headaches, an inability to sleep, muscle weakness, profuse sweating, body odor, enlarged sebaceous glands, which are glands that produce oils in the skin, thickened skin, skin tags, which are noncancerous growths. Acromegaly can start any time after puberty. However, it occurs more often in middle age.

* Addison disease, adrenal hormones disorder with symptoms of weight loss, muscle weakness, fatigue, low blood pressure, and skin color changes.

* Adenoma, benign tumor of glandular origin which may become malignant.

* Adenomatous polyposis coli, a mutation in the APC gene which may result in colorectal cancer.[

* Adenomyosis is a medical condition characterized by the presence of ectopic glandular tissue found in the muscular wall of the uterus (myometria). The term adenomyosis is derived from the Greek terms adeno- (meaning gland), myo- (meaning muscle), and -osis (meaning condition). Previously named as endometriosis interna, but adenomyosis differs from endometriosis and these two diseases represent two separate entities. They are found together in many cases. It usually refers to ectopic endometrial tissue (the inner lining of the uterus) within the myometrium (the thick, muscular layer of the uterus). The term "adenomyometritis" specifically implies involvement of the uterus. The condition is typically found in women between the ages of 35 and 50 but can also be present in younger women. Patients with adenomyosis often present with painful and/or profuse menses (dysmenorrhea & menorrhagia, respectively). Other possible symptoms are pain during sexual intercourse, chronic pelvic pain and irritation of the urinary bladder.

* Adenovirus infections most commonly cause illness of the respiratory system; however, depending on the infecting serotype, they may also cause various other illnesses and presentations. Apart from respiratory involvement, illnesses and presentations of adenovirus include gastroenteritis, conjunctivitis, cystitis, and rash illness. Symptoms of respiratory illness caused by adenovirus infection range from the common cold syndrome to pneumonia, croup, and bronchitis. Patients with compromised immune systems are especially susceptible to severe complications of adenovirus infection. Acute respiratory disease (ARD), first recognized among military recruits during World War II, can be caused by adenovirus infections during conditions of crowding and stress. Pharyngoconjunctival fever is a specific presentation of adenovirus infection, manifested as: high fever that lasts 4-5 days, pharyngitis (sore throat), conjunctivitis (inflamed eyes, usually without pus formation like pink eye), enlargement of the lymph nodes of the neck, headache, malaise, and weakness. It usually occurs in the age group 5-18. It is often found in summer camps and during the spring and fall in schools. Although epidemiologic characteristics of the adenoviruses vary by type, all are transmitted by direct contact, fecal-oral transmission, and occasionally waterborne transmission. Several adenoviruses, including Ad5, Ad9, Ad31, Ad36, Ad37, and SMAM1, have at least some evidence of causation of obesity in animals, adipogenesis in cells, and/or association with human obesity. Adenovirus can cause severe necrotizing pneumonia in which all or part of a lung has increased translucency radiographically, which is called Swyer-James Syndrome. Severe adenovirus pneumonia also may result in bronchiolitis obliterans, a subacute inflammatory process in which the small airways are replaced by scar tissue, resulting in a reduction in lung volume and lung compliance.

* Adhesions pelvic, bands of scar tissue that attach to organs in the pelvis.

* Adiposis dolorosa, painful folds of fatty (adipose) tissue or the growth of multiple noncancerous (benign) fatty tumors called lipomas.

* Adnexitis, inflammatory disease that affects the fallopian tubes and ovaries, or the uterus.

* Adrenoleukodystrophy, several closely related inherited disorders that disrupt the breakdown (metabolism) of certain fats, and their accumulation in tissues like myelin disrupt health.

* African trypanosomiasis or sleeping sickness is a parasitic disease of humans and other animals. It is caused by protozoa of the species Trypanosoma brucei. There are two types that infect humans, Trypanosoma brucei gambiense (T.b.g) and Trypanosoma brucei rhodesiense (T.b.r.). T.b.g causes over 98% of reported cases. Both are usually transmitted by the bite of an infected tsetse fly and are most common in rural areas.

* Agammaglobulinemia, a type of primary immune deficiency disease characterized by absence of gamma globulins.

* Aganglionosis colonic, when part or all of the large intestine or antecedent parts of the gastrointestinal tract have no nerves and therefore cannot function.

* Aicardi syndrome is an extremely rare genetic disorder. Almost all people with Aicardi syndrome are females. Individuals with Aicardi syndrome have agenesis of the corpus callosum, chorioretinal lacunae and seizures. Agenesis of the corpus callosum means that the structure that connects the right half of the brain with the left half of the brain (corpus callosum) does not develop normally. Chorioretinal lacunae are small holes in the retina (back part of the eye). These are only visible to a doctor using a device to look into the back of the eye. It is very unusual (maybe impossible) to have Aicardi syndrome without having chorioretinal lacunae. The seizures can be of different types. Infants with Aicardi syndrome usually have a type of seizures known as "infantile spasms". These are single jerks of the whole body. They may happen many times a day. Infants are usually awake during infantile spasms. These look different from the more common type of seizures known as "generalized tonic-clonic" seizures. Generalized tonic-clonic seizures are rhythmic jerking of arms and legs. People often black out during this type of seizure. Children with Aicardi syndrome usually grow out of infantile spasms and then have generalized tonic-clonic or other types of seizures. Other parts of the brain also do not develop normally. These brain malformations cause frequent seizures and intellectual disability.

* AIDS-HIV or human immunodeficiency virus infection and acquired immune deficiency syndrome is a spectrum of conditions caused by infection with the human immunodeficiency virus (HIV). Following initial infection, a person may not notice any symptoms or may experience a brief period of influenza-like illness.Typically, this is followed by a prolonged period with no symptoms. As the infection progresses, it interferes more with the immune system, increasing the risk of common infections like tuberculosis, as well as other opportunistic infections, and tumors that rarely affect people who have working immune systems. These late symptoms of infection are referred to as AIDS. This stage is often also associated with weight loss.

* Aldosteronism, abnormality of the body's electrolyte balance, caused by excessive secretion of aldosterone by the adrenal cortex.

* Alexander disease is a rare disorder of the nervous system. It is one of a group of disorders, called leukodystrophies, that involve the destruction of myelin. Myelin is the fatty covering that insulates nerve fibers and promotes the rapid transmission of nerve impulses. If myelin is not properly maintained, the transmission of nerve impulses could be disrupted. As myelin deteriorates in leukodystrophies such as Alexander disease, nervous system functions are impaired. Most cases of Alexander disease begin before age 2 and are described as the infantile form. Signs and symptoms of the infantile form typically include an enlarged brain and head size (megalencephaly), seizures, stiffness in the arms and/or legs (spasticity), intellectual disability, and developmental delay. Less frequently, onset occurs later in childhood (the juvenile form) or in adulthood. Common problems in juvenile and adult forms of Alexander disease include speech abnormalities, swallowing difficulties, seizures, and poor coordination (ataxia). Rarely, a neonatal form of Alexander disease occurs within the first month of life and is associated with severe intellectual disability and developmental delay, a buildup of fluid in the brain (hydrocephalus), and seizures. Alexander disease is also characterized by abnormal protein deposits known as Rosenthal fibers. These deposits are found in specialized cells called astroglial cells, which support and nourish other cells in the brain and spinal cord (central nervous system).

* Alkalosis is the result of a process reducing hydrogen ion concentration of arterial blood plasma (alkalemia). In contrast to acidemia (serum pH 7.35 or lower), alkalemia occurs when the serum pH is higher than normal (7.45 or higher). Alkalosis is usually divided into the categories of respiratory alkalosis and metabolic alkalosis or a combined alkalosis. Respiratory alkalosis is caused by hyperventilation, resulting in a loss of carbon dioxide. Hyperventilation-induced alkalosis can be seen in several deadly central nervous system diseases such as strokes or Rett syndrome. Metabolic alkalosis can be caused by repeated vomiting, resulting in a loss of hydrochloric acid within the stomach content. Severe dehydration, and the consumption of alkali are other causes. It can also be caused by administration of diuretics and endocrine disorders such as Cushing's syndrome. As respiratory acidosis often accompanies the compensation for metabolic alkalosis, and vice versa, a delicate balance is created between these two conditions. Metabolic alkalosis is usually accompanied with low blood potassium level, causing, e.g., muscular weakness, muscle pain, and muscle cramps (from disturbed function of the skeletal muscles), and muscle spasms (from disturbed function of smooth muscles). It may also cause low blood calcium levels.

* Alkaptonuria, genetic defect that makes the body unable to properly break down tyrosine and phenylalanine, causing the accumulation and eventual excretion of alkapton.

* All diabetes comprehensive -type-1, type-2, Onset. Diabetes is a group of metabolic diseases in which there are high blood sugar levels over a prolonged period. Symptoms of high blood sugar include frequent urination, increased thirst, and increased hunger. If left untreated, diabetes can cause many complications. Acute complications can include diabetic ketoacidosis, nonketotic hyperosmolar coma, or death. Serious long-term complications include heart disease, stroke, chronic kidney failure, foot ulcers, and damage to the eyes. Diabetes is due to either the pancreas not producing enough insulin or the cells of the body not responding properly to the insulin produced.

* Allergic granulomatous angiitis (AGA) is a rare disease, which varies in clinical manifestation. The clinical course includes three stages. In the first stage most of the patients show symptoms of respiratory disease, 82 per cent of the patients has asthma and 88 per cent chronic rhinitis. In the second stage there is blood eosinophilia and in the last stage involvement of multiple organs.

* Alpha 1 antitrypsin deficiency, condition in which the body does not make enough of a protein that protects the lungs and liver from damage.

* Alpha-mannosidosis is an autosomal recessive lysosomal storage disease characterized by mental retardation, coarse facial features, skeletal abnormalities, hearing impairment, neurologic motor problems, and immune deficiency. Expression of the disease varies considerably, and there is a wide spectrum of clinical findings and severity. Affected children are often normal at birth and during early development. They present in early childhood with delayed psychomotor development, delayed speech, and hearing loss. Additional features include large head with prominent forehead, rounded eyebrows, flattened nasal bridge, macroglossia, widely spaced teeth, dysostosis multiplex, and motor impairment. Lysosomes are particles bound in membranes within cells that function as the primary digestive units within cells. Enzymes within lysosomes break down or digest particular nutrients, such as complex molecules composed of a sugar attached to a protein (glycoproteins). Low levels or inactivity of the alpha-mannosidase enzyme leads to the abnormal accumulation these compounds in the cells of affected individuals with unwanted consequences.

* Altitude sickness, pathological effect of high altitude on humans.

* Amblyopia, decrease in vision in one or both eyes with no apparent structural abnormality.

* Amniotic band syndrome, congenital birth defects caused by the entrapment of fetal body parts by fibrous amniotic bands during pregnancy.

* Amyloidosis, group of diseases resulting from abnormal deposition of certain proteins (amyloids) in various bodily areas.

* An acoustic neuroma or vestibular schwannoma is a benign primary intracranial tumor of the myelin-forming cells of the vestibulocochlear nerve (8th cranial nerve). A type of schwannoma, this tumor arises from the Schwann cells responsible for the myelin sheath that helps keep peripheral nerves insulated. The primary symptoms of vestibular schwannoma are unexplained unilateral hearing loss and tinnitus, pressure in the ear (plugged or "full" ear) and vestibular (disequilibrium) symptoms. Treatment by surgery or radiation often results in substantial or complete hearing loss in the affected ear. Observation (non-treatment) over time also usually results in hearing loss in the affected ear. Larger tumors can press on the trigeminal nerve (CN V), causing facial numbness and tingling - constantly or intermittently. Paradoxically, the facial nerve (CN VII) is rarely involved pre-operatively; although it may be compressed by the tumor, it is unusual for patients to experience weakness or paralysis of the face.

* An inflammatory response or sepsis syndrome represents a systemic response to infection and is defined as either hypothermia or hyperthermia, tachycardia, tachypnea, and evidence of inadequate organ function or perfusion manifested by at least one of the following: altered mental status, hypoxemia, acidosis, oliguria, or disseminated intravascular coagulation.

* An ovarian cyst is a fluid-filled sac within the ovary. Often they cause no symptoms. Occasionally they may produce bloating, lower abdominal pain, or lower back pain. If the cyst either breaks open or causes twisting of the ovary severe pain may occur. This may result in vomiting or feeling faint. The majority of cysts are, however, harmless. Most ovarian cysts are related to ovulation being either follicular cysts or corpus luteum cysts. Other types include cysts due to endometriosis, dermoid cysts, and cystadenomas. Many small cysts occur in both ovaries in polycystic ovarian syndrome. Pelvic inflammatory disease may also result in cysts. Rarely cysts may be a form of ovarian cancer. Some or all of the following symptoms may be present, though it is possible not to experience any symptoms. Abdominal pain. Dull aching pain within the abdomen or pelvis, especially during intercourse. Uterine bleeding. Pain during or shortly after beginning or end of menstrual period; irregular periods, or abnormal uterine bleeding or spotting. Fullness, heaviness, pressure, swelling, or bloating in the abdomen. When a cyst ruptures from the ovary, there may be sudden and sharp pain in the lower abdomen on one side. Change in frequency or ease of urination (such as inability to fully empty the bladder), or difficulty with bowel movements due to pressure on adjacent pelvic anatomy. Constitutional symptoms such as fatigue, headaches. Nausea or vomiting. Weight gain. Other symptoms may depend on the cause of the cysts. Symptoms that may occur if the cause of the cysts is polycystic ovarian syndrome (PCOS) may include increased facial hair or body hair, acne, obesity and infertility. If the cause is endometriosis, then periods may be heavy, and intercourse painful. The effect of cysts not related to PCOS on fertility is unclear.

* An umbilical hernia is a health condition where the abdominal wall behind the navel is damaged. It may cause the navel to bulge outwards - the bulge consisting of abdominal fat from the greater omentum or occasionally parts of the small intestine. The bulge can often be pressed back through the hole in the abdominal wall, and may "pop out" when coughing or otherwise acting to increase intra-abdominal pressure. A hernia is present at the site of the umbilicus (commonly called a navel, or belly button) in the newborn; although sometimes quite large, these hernias tend to resolve without any treatment by around the age of 2-3 years. Obstruction and strangulation of the hernia is rare because the underlying defect in the abdominal wall is larger than in an inguinal hernia of the newborn. The size of the base of the herniated tissue is inversely correlated with risk of strangulation (i.e. narrow base is more likely to strangulate). Babies are prone to this malformation because of the process during fetal development by which the abdominal organs form outside the abdominal cavity, later returning into it through an opening which will become the umbilicus. Hernias may be asymptomatic and present only as a bulge of the umbilicus. Symptoms may develop when the contracting abdominal wall causes pressure on the hernia contents. This results in abdominal pain or discomfort. These symptoms may be worsened with lifting and straining.

* Anemia is usually defined as a decrease in the amount of red blood cells (RBCs) or hemoglobin in the blood. It can also be defined as a lowered ability of the blood to carry oxygen. When anemia comes on slowly, the symptoms are often vague and may include: feeling tired, weakness, shortness of breath or a poor ability to exercise. Anemia that comes on quickly often has greater symptoms, which may include: confusion, feeling like one is going to pass out, loss of consciousness, or increased thirst. Anemia must be significant before a person becomes noticeably pale. Additional symptoms may occur depending on the underlying cause. It is more common in females than males, among children, during pregnancy, and in the elderly. Anemia goes undetected in many people and symptoms can be minor. The symptoms can be related to an underlying cause or the anemia itself. Most commonly, people with anemia report feelings of weakness, or fatigue, general malaise, and sometimes poor concentration. They may also report dyspnea (shortness of breath) on exertion. In very severe anemia, the body may compensate for the lack of oxygen-carrying capability of the blood by increasing cardiac output.

* Aneurysm, a sac formed by localized dilatation of the wall of an artery or a vein.

* Angina pectoris, chest pain due to an inadequate supply of oxygen to the heart muscle.

* Angiofibroma, focal growth of vascular and collagen structures normally found in the skin.

* Angiokeratoma corporis diffusum is a clinical variant of angiokeratoma that is typically caused by a deficiency of the lysosomal enzyme alpha-galactosidase. Angiokeratomas are hyperkeratotic papules that are characterized histologically by superficial ectatic vessels with epidermal proliferation. They occur clinically as 1 of 5 different subtypes. Angiokeratoma corporis diffusum (ACD) occurs most typically on the lower region of the trunk, buttocks, and thighs. Angiokeratoma corporis diffusum occurs in several other diseases related to deficiencies of enzymes that are involved in the metabolism of glycoproteins, including fucosidosis, sialidosis, mannosidosis, GM1 gangliosidosis, and Kanzaki disease. Very rarely, ACD has been reported in patients with normal enzyme activity. Angiokeratoma corporis diffusum (Fabry disease) is rare; the estimated incidence is 1 case per 40,000 population. Most occurrences of angiokeratoma corporis diffusum (Fabry disease) are in whites. Because angiokeratoma corporis diffusum (Fabry disease) is an X-linked recessive disease, only men are fully afflicted. Heterozygous women, in addition to transmitting the condition, may develop symptoms. In males, signs and symptoms of angiokeratoma corporis diffusum (Fabry disease) begin in late childhood or adolescence.

* Angiolymphoid, benign but potentially disfiguring vascular proliferative disease presenting as collections of papules, nodules or plaques, most commonly in the head and neck area.

* Angioma is a benign tumor derived from cells of the vascular or lymphatic vessel walls (endothelium) or derived from cells of the tissues surrounding these vessels.

* Anhidrosis is the complete absence of sweating, while hypohidrosis is weating less than normal. If the human body cannot sweat properly it cannot cool itself, which is potentially harmful. Sweating allows heat to be released from the body. If the lack of sweating affects a small area of the body, it is not usually dangerous. However, overall anhidrosis or hypohidrosis can result in overheating and eventually heatstroke, a potentially life-threatening condition.

* Aniridia is the absence of the iris, usually involving both eyes. It can be congenital or caused by a penetrant injury. Isolated aniridia is a congenital disorder which is not limited to a defect in iris development, but is a panocular condition with macular and optic nerve hypoplasia, cataract, and corneal changes. Vision may be severely compromised and the disorder is frequently associated with a number of ocular complications: nystagmus, amblyopia, buphthalmos, and cataract. Aniridia in some individuals occurs as part of a syndrome, such as WAGR syndrome (kidney nephroblastoma (Wilms tumour), genitourinary anomalies and intellectual disability), or Gillespie syndrome (cerebellar ataxia).

* Anisakiasis is a human parasitic infection of the gastrointestinal tract caused by the consumption of raw or undercooked seafood containing larvae of the nematode Anisakis simplex. Within a few hours of ingestion, the parasitic worm tries to burrow though the intestinal wall, but since it cannot penetrate it, it gets stuck and dies. The presence of the parasite triggers an immune response; immune cells surround the worms, forming a ball-like structure that can block the digestive system, causing severe abdominal pain, malnutrition and vomiting. Occasionally, the larvae are regurgitated. If the larvae pass into the bowel or large intestine, a severe eosinophilic granulomatous response may also occur one to two weeks following infection, causing symptoms mimicking Crohn's disease. Anisakiasis can be easily prevented by adequate cooking at temperatures greater than 60 degree C or freezing. Even when the fish is thoroughly cooked, Anisakis larvae pose a health risk to humans. Anisakids and related species release a number of biochemicals into the surrounding tissues when they infect a fish. Acute allergic manifestations, such as urticaria and anaphylaxis, may occur with or without accompanying gastrointestinal symptoms. Distribution is worldwide but there is a higher incidence in areas where raw fish is eaten - for example, Japan, the Pacific coast of South America, and the Netherlands.

* Anisocoria is a condition characterized by an unequal size of the eyes' pupils. Affecting 20% of the population, it can be an entirely harmless condition or a symptom of more serious medical problems. Anisocoria which is greater in dim light suggests Horner's syndrome or mechanical anisocoria. In Horner's syndrome sympathetic fibers have a defect, therefore the pupil of the involved eye will not dilate in darkness. Anisocoria which is greater in bright light suggests Adie tonic pupil, pharmacologic dilation, oculomotor nerve palsy, or damaged iris. Some of the causes of anisocoria are life-threatening, including Horner's syndrome (which may be due to carotid dissection) and oculomotor nerve palsy (due to a brain aneurysm, uncal herniation, or head trauma). Anisocoria is usually a benign finding, unaccompanied by other symptoms (physiological anisocoria). It should be considered an emergency if a patient develops acute onset anisocoria. These cases may be due to brain mass lesions which cause oculomotor nerve palsy. Anisocoria in the presence of confusion, decreased mental status, severe headache, or other neurological symptoms can forewarn a neurosurgical emergency. This is because a hemorrhage, tumor or another intracranial mass can enlarge to a size where the third cranial nerve (CN III) is compressed, which results in uninhibited dilatation of the pupil on the same side as the lesion.

* Anophthalmos or anophthalmia, is the medical term for the absence of one or both eyes. Both the globe (human eye) and the ocular tissue are missing from the orbit. The absence of the eye will cause a small bony orbit, a constricted mucosal socket, short eyelids, reduced palpebral fissure and malar prominence. Anophthalmia is an extremely rare disease and is mostly rooted in genetic abnormalities. It can also be associated with other syndromes. Anophthalmia has been reported to be present in 3 out of every 100,000 births. Many instances of anophthalmia also occur with microphthalmia. A recent study in the UK indicated that anophthalmia and microphthalmia had a combined average of 1 in every 10,000 births.

* Antiphosholipid syndrome, autoimmune production of antibodies against phospholipid (aPL), a cell membrane substance.

* Antithrombin III deficiency is a blood disorder characterized by the tendency to form clots in the veins (thrombosis). An inherited tendency to thrombosis is known as thrombophilia. Antithrombin is a substance in the blood that limits the blood's ability to clot (coagulation). In people with congenital antithrombin deficiency, there is a reduced amount of this substance in the blood due to a genetic abnormality. Antithrombin deficiency may also be acquired. The type of blood clots seen in individuals with this condition are typically clots that form in the deep veins of the leg (deep vein thrombosis or DVT) and clots that lodge in the lungs (pulmonary embolism or PE). Approximately 50% of individuals with antithrombin deficiency will develop one or more clots in their lifetime, usually after adolescence. Factors that may increase the likelihood of clotting include pregnancy, the use of oral contraceptives, surgery, increasing age, and a lack of movement. Blood clots can cause swelling, pain, or shortness of breath. Other symptoms of blood clots include dizziness, headache, changes in vision, lack of concentration, confusion, slurred speech, numbness, tingling, and weakness in the arms or legs. Blood clots may become life threatening if not recognized and treated early.

* Aortic valve stenosis - or aortic stenosis - occurs when the heart's aortic valve narrows. This narrowing prevents the valve from opening fully, which obstructs blood flow from the heart into the aorta and onward to the rest of the body. When the aortic valve is obstructed, the heart needs to work harder to pump blood. Eventually, this extra work limits the amount of blood it can pump and may weaken the heart muscle. Aortic stenosis is the most common valvular heart disease in the developed world. Aortic stenosis is most commonly caused by age-related progressive calcification (>50% of cases) with a mean age of 65 to 70 years. Another major cause of aortic stenosis is the calcification of a congenital bicuspid aortic valve[14] (30-40% of cases) typically presenting earlier, in those aged 40+ to 50+.

* Aortitis is the inflammation of the aortic wall. The disorder is potentially life-threatening and rare. Aortitis is most common in people 10 to 40 years of age. This inflammation has a number of possible causes, including trauma, viral or bacterial infections (notably, syphilis), and certain immune disorders and connective tissue diseases. Aortitis is most commonly seen in patients with syphilis, autoimmune vasculitis (giant cell arteritis, Takayasu's arteritis), polymyalgia rheumatica, and rheumatoid arthritis. IgG4-related disease is a more recently identified cause of aortitis and periaortitis. There is a wide range of symptoms that are dependent on the location of the aortic inflammation or associated disorder. A few symptoms recognized in patients are fever, chills, muscle pains, and malaise (general discomfort). In addition, hypertension (high blood pressure) may occur. Hypertension happens when the renal artery narrows and elasticity of the aorta and branches decrease. If untreated, has three distinct phases. The first is a prepulseless inflammatory stage with nonspecific symptoms such as fatigue, arthralgias, and low-grade fevers. Phase two includes vascular inflammation with pain secondary to the condition, along with tenderness to palpation over the site. The last phase includes symptoms of ischemia and pain associated with the use of limbs. Limbs are also cool and clammy in this stage.

* Aplastic anemia, a condition where bone marrow does not produce sufficient new cells to replenish blood cells.

* Apnea, temporary absence or cessation of breathing.

* Appendicitis, water fast for 48 hours.

* Aprosencephaly is an extremely rare anomaly of fetal cerebral development where the derivatives of the telencephalon as well as the diancephalon in the brain are absent or dysplastic, while more caudal structures are normal or mildly deformed. Other signs may include oculo-facial anomalies (i.e. ocular hypotelorism or cyclopia, malformation/absence of nasal structures, cleft lip), preaxial limb defects (i.e. hypoplastic hands, absent halluces) and various other anomalies including ambiguous genitalia, imperforate anus, and vertebral anomalies. It falls under the aprosencephaly / atelencephaly spectrum (AAS). Atelencephaly is also called aprosencephaly with open cranium.

* Arachnoid cysts are cysts, not tumors. They are fluid-filled sacs that develop between the brain/spinal cord and a membrane called the arachnoid membrane, one of three membranes that cover the brain and spinal cord. If the arachnoid membrane splits or tears, it can trap cerebrospinal fluid - the fluid that surrounds and protects the brain from damage. The cysts grow as more fluid becomes trapped. Symptoms vary depending on the size and location of the arachnoid cyst and can go undetected for years. Small cysts - and even many large ones - often exist without causing symptoms. However, if the cyst grows large enough it can put pressure on surrounding parts of the brain and spinal cord, resulting in the onset of symptoms. Most common symptoms include: Headaches. Nausea and/or vomiting. Seizures. Behavioral changes. Hearing and vision problems. Vertigo. Balance problems while walking or standing. Weakness or paralysis on one side of the body.

* Arachnoiditis, caused by inflammation of the arachnoid, one of the membranes that surround and protect the nerves of the central nervous system.

* Arbovirus is a term used to refer to a group of viruses that are transmitted by arthropod vectors. Symptoms of arbovirus infection generally occur 3 -15 days after exposure to the virus and last 3 or 4 days. The most common clinical features of infection are fever, headache, and malaise, but encephalitis and hemorrhagic fever may also occur. The word tibovirus is sometimes used to describe viruses transmitted by ticks, a superorder within the arthropods. Arboviruses maintain themselves in nature by going through a cycle between a host, an organism that carries the virus, and a vector, an organism that carries and transmits the virus to other organisms. Person-to-person transmission of arboviruses is not common, but can occur. Blood transfusions, organ transplantation, and the use of blood products can transmit arboviruses if the virus is present in the donor's blood or organs.

* Arnold-Chiari malformations (CMs) are structural defects in the cerebellum. They consist of a downward displacement of the cerebellar tonsils through the foramen magnum (the opening at the base of the skull), sometimes causing non-communicating hydrocephalus as a result of obstruction of cerebrospinal fluid (CSF) outflow. The cerebrospinal fluid outflow is caused by phase difference in outflow and influx of blood in the vasculature of the brain. CMs can cause headaches, difficulty focusing, concentrating, and thinking, as well as difficulty coping (with what most consider and experience as normal, everyday life), fatigue, muscle weakness, difficulty swallowing (sometimes accompanied by gagging), choking and vomiting, dizziness, nausea, tinnitus, impaired coordination, neck pain, unsteady gait (problems with balance), poor hand coordination (fine motor skills), numbness and tingling of the hands and feet, and speech problems (such as hoarseness).

* Arrhythmogenic right ventricular dysplasia (ARVD) is an inherited heart disease. ARVD is caused by genetic defects of the parts of heart muscle (also called myocardium or cardiac muscle) known as desmosomes, areas on the surface of heart muscle cells which link the cells together. The desmosomes are composed of several proteins, and many of those proteins can have harmful mutations. The disease is a type of nonischemic cardiomyopathy that involves primarily the right ventricle. It is characterized by hypokinetic areas involving the free wall of the right ventricle, with fibrofatty replacement of the right ventricular myocardium, with associated arrhythmias originating in the right ventricle. ARVD can be found in association with diffuse palmoplantar keratoderma, and woolly hair, in an autosomal recessive condition called Naxos disease, because this genetic abnormality can also affect the integrity of the superficial layers of the skin most exposed to pressure stress. ARVC/D is an important cause of ventricular arrhythmias in children and young adults. It is seen predominantly in males, and 30 - 50% of cases have a familial distribution. Up to 80% of individuals with ARVD present have symptoms like syncope and dyspnea.The remainder frequently present with palpitations or other symptoms due to right ventricular outflow tract (RVOT) tachycardia (a type of monomorphic ventricular tachycardia). Symptoms are usually exercise-related. In populations where hypertrophic cardiomyopathy is screened out prior to involvement in competitive athletics, it is a common cause of sudden cardiac death. The first clinical signs of ARVD are usually during adolescence. However, signs of ARVD have been demonstrated in infants.

* Arteriosclerosis, hardening (and loss of elasticity) of medium or large arteries.

* Arteriovenous malformation (AVM) is an abnormal connection between arteries and veins, bypassing the capillary system. This vascular anomaly is widely known because of its occurrence in the central nervous system, but can appear in any location. Although many AVMs are asymptomatic, they can cause intense pain or bleeding or lead to other serious medical problems.

* Arteritis temporal, inflammation and damage to the blood vessels that supply blood to the head.

* Arthritis reactive is classified as an autoimmune condition that develops in response to an infection in another part of the body (cross-reactivity). Coming into contact with bacteria and developing an infection can trigger the disease. The manifestations of reactive arthritis include the following triad of symptoms: an inflammatory arthritis of large joints, inflammation of the eyes in the form of conjunctivitis or uveitis, and urethritis in men or cervicitis in women. Arthritis occurring alone following sexual exposure or enteric infection is also known as reactive arthritis. Patients can also present with mucocutaneous lesions, as well as psoriasis-like skin lesions such as circinate balanitis, and keratoderma blennorrhagicum. Enthesitis can involve the Achilles tendon resulting in heel pain. Not all affected persons have all the manifestations. The clinical pattern of reactive arthritis commonly consists of an inflammation of fewer than five joints which often includes the knee or sacroiliac joint. The arthritis may be "additive" (more joints become inflamed in addition to the primarily affected one) or "migratory" (new joints become inflamed after the initially inflamed site has already improved).

* Arthrogryposis, congenital disorder marked by generalized stiffness of the joints, often accompanied by muscle and nerve degeneration, resulting in severely impaired mobility of the limbs.

* Asbestosis is a chronic inflammatory and scarring disease affecting the tissue of the lungs. People with the condition may experience severe shortness of breath and are at an increased risk for certain cancers, including lung cancer and, less commonly, mesothelioma. Asbestosis specifically refers to fibrosis within the lung tissue from asbestos, and not scarring around the outside of the lungs.

* Ascariasis is a disease caused by the parasitic roundworm Ascaris lumbricoides. Infections have no symptoms in more than 85% of cases, especially if the number of worms is small. Symptoms increase with the number of worms present and may include shortness of breath and fever in the beginning of the disease. These may be followed by symptoms of abdominal swelling, abdominal pain, and diarrhea. Children are most commonly affected, and in this age group the infection may also cause poor weight gain, malnutrition, and learning problems.

* Ascorbic acid deficiency, or deficiency of vitamin C, which is required for the synthesis of collagen in humans.

* Asthma plus allergies comprehensive.

* Astigmatism is a type of refractive error in which the eye does not focus light evenly on the retina. This results in distorted or blurred vision at all distances. Other symptoms can include eyestrain, headaches, and trouble driving at night. If it occurs early in life it can result in amblyopia. Although astigmatism may be asymptomatic, higher degrees of an astigmatism may cause symptoms such as blurry vision, squinting, eye strain, fatigue, or headaches. Some research has pointed to the link between astigmatism and higher prevalence of migraine headaches.

* Astrocytoma brain tumors are a form of glioma with star-shaped cells. They often grow very slowly or not at all for long periods of time. General symptoms of an astrocytoma tumor are a result of growing pressure inside the skull. These symptoms include headache, vomiting and mental status changes. Other symptoms, such as drowsiness, lethargy, obtuseness, personality changes, disordered conduct and impaired mental faculties show up early in about one out of every four patients with malignant brain tumors. In young children, the growing pressure of an astrocytoma tumor inside the skull may enlarge the head. Changes (such as swelling) may be observed in the back of the eye, where the blind spot is. Usually there are no changes in temperature, blood pressure, pulse or respiratory rates except just before death. Seizures are more common with meningiomas, slow-growing astrocytomas and oligodendrogliomas than with malignant gliomas. Symptoms of an astrocytoma tumor vary depending on what part of the brain (or which glands or nerves) are affected by the tumor. Sometimes the nature of the seizures can help determine the location of the brain tumor.

* Ataxia-telangiectasia is a rare inherited disorder that affects the nervous system, immune system, and other body systems. This disorder is characterized by progressive difficulty with coordinating movements (ataxia) beginning in early childhood, usually before age 5. The movement problems typically cause people to require wheelchair assistance by adolescence. People with this disorder also have slurred speech and trouble moving their eyes to look side-to-side (oculomotor apraxia). Small clusters of enlarged blood vessels called telangiectases, which occur in the eyes and on the surface of the skin, are also characteristic of this condition. Affected individuals tend to have high amounts of a protein called alpha-fetoprotein (AFP) in their blood. The level of this protein is normally increased in the bloodstream of pregnant women, but it is unknown why individuals with ataxia-telangiectasia have elevated AFP or what effects it has in these individuals. People with ataxia-telangiectasia often have a weakened immune system, and many develop chronic lung infections. They also have an increased risk of developing cancer, particularly cancer of blood-forming cells (leukemia) and cancer of immune system cells (lymphoma). Affected individuals are very sensitive to the effects of radiation exposure, including medical x-rays. The life expectancy of people with ataxia-telangiectasia varies greatly, but affected individuals typically live into early adulthood.