Treatment combo Sessions of Modern Medicine in Digital format

Treatment combo Sessions of Modern Medicine in Digital format - G

The frequencies used in these sessions are based upon Rife sets for resonant therapy devices masked in Algorithmic piano music.

More information regarding the items in the list is given below the list.

List of Treatment combo Sessions of Modern Medicine in Digital format for problems/products available at us for just Rs. 1,000/- for any 5 sessions from any one or multiple Treatment combo Sessions for 30 doses per session (2 times a day for 15 days) in max 15 days.

1) Galactosemias
2) Gallbladder Diseases
3) Gallbladder Inflammation
4) Gangrene
5) Gastric Stasis
6) Gastritis
7) Gastroenteritis
8) Gastroesophageal Reflux
9) Gastrointestinal Disease
10) Gastrointestinal Hemorrhage
11) Gastrointestinal Post-Surgery
12) Gastrointestinal Stromal Tumors
13) Gastroschisis
14) General Fibrosis Syndrome
15) General Prophylaxis
16) Genital Diseases-Female
17) Genital Diseases-Male
18) Gerstmann-Straussler Syndrome
19) Giant Lymph Node Hyperplasia
20) Giardiasis
21) Gilbert Disease
22) Gingivitis
23) Glaucoma
24) Glioblastoma
25) Glioma
26) Glomerulonephritis
27) Glossopharyngeal Neuralgia
28) Glutaric Acidemia
29) Glycogenosis
30) Glycoprotein Syndrome
31) Gonadal Disorders
32) Gonorrhea
33) Goodpasture Syndrome
34) Gout
35) Granuloma
36) Granuloma Annulare
37) Granuloma Pseudopyogenic
38) Granuloma Venereum
39) Guinea Worm Infection
40) Gynecomastia
41) Gyrate Atrophy

* Galactosemias, condition in which the body is unable to use (metabolize) the simple sugar galactose.

* Gallbladder inflammation or cholecystitis symptoms include right upper abdominal pain, nausea, vomiting, and occasionally fever. Often acute cholecytitis is proceeded by gallbladder attacks. The pain, however, lasts longer than is typical in a gallbladder attack. Without appropriate treatment recurrent episodes of cholecystitis are common. Acute cholecystitis may be complicated by gallstone pancreatitis, common bile duct stones, or inflammation of the common bile duct. More than 90% of the time acute cholecystitis is from blockage of the bile duct by a gallstone. Risk factors for gallstones include birth control pills, pregnancy, a family history of gallstones, obesity, diabetes, liver disease, or rapid weight loss. Occasionally acute cholecystitis occur as a result of vasculitis, chemotherapy, or during recovery from major trauma or burns. Most people with gallstones do not have symptoms. When a gallstone becomes intermittently lodged in the cystic duct, they suffer from biliary colic. Biliary colic is abdominal pain in the right upper quadrant or epigastrium that is usually episodic, occurs after eating greasy or fatty foods, and leads to nausea and/or vomiting. People who suffer from cholecystitis most commonly initially have symptoms of biliary colic before developing cholecystitis. The symptoms of cholecystitis are similar to biliary colic but the pain becomes more severe and constant. Nausea is common and vomiting occurs in 75% of people with cholecystitis. In addition to abdominal pain, right shoulder pain can be present. On physical examination, fever is common. A gallbladder with cholecystitis is almost always tender to touch. Because of the inflammation, its size can be felt from the outside of the body in 25-50% of people with cholecystitis. Pain with deep inspiration leading to termination of the breath while pressing on the right upper quadrant of the abdomen usually causes pain (Murphy's sign). Murphy's sign is sensitive, but not specific for cholecystitis. Jaundice may occur but is usually mild, and if severe, suggests another cause of symptoms such as choledocholithiasis. Elderly patients and those with diabetes, chronic illness, or who are immunocompromised may have vague symptoms that may not include fever or localized tenderness.

* Gastric stasis, delayed gastric emptying in the absence of a mechanical obstruction, with symptoms such as nausea and vomiting.
* Gastritis, inflammation in the protective lining of the stomach.

* Gastroenteritis, infection or irritation of the digestive tract, particularly the stomach and intestine.

* Gastrointestinal bleeding (GI bleed), also known as gastrointestinal hemorrhage, is all forms of bleeding in the gastrointestinal tract, from the mouth to the rectum. When there is significant blood loss over a short time, symptoms may include vomiting red blood, vomiting black blood, bloody stool, or black stool. Small amounts of bleeding over a long time may cause iron-deficiency anemia resulting in feeling tired or heart-related chest pain. Other symptoms may include abdominal pain, shortness of breath, pale skin, or passing out. Sometimes in those with small amounts of bleeding no symptoms may be present.

* Gastrointestinal stromal tumors (GISTs) are the most common mesenchymal neoplasms of the gastrointestinal tract. GISTs arise in the smooth muscle pacemaker interstitial cell of Cajal, or similar cells. Most (66%) occur in the stomach and gastric GISTs have a lower malignant potential than tumors found elsewhere in the GI tract. GISTs may present with trouble swallowing, gastrointestinal bleeding, or metastases (mainly in the liver). Intestinal obstruction is rare, due to the tumor's outward pattern of growth. Often, there is a history of vague abdominal pain or discomfort, and the tumor has become rather large by time the diagnosis is made. GISTs are tumors of connective tissue, i.e. sarcomas; unlike most gastrointestinal tumors, they are nonepithelial. About 70% occur in the stomach, 20% in the small intestine and less than 10% in the esophagus. Small tumors are generally benign, especially when cell division rate is slow, but large tumors disseminate to the liver, omentum and peritoneal cavity. They rarely occur in other abdominal organs. GISTs occur in 10-20 per one million people. The true incidence might be higher, as novel laboratory methods are much more sensitive in diagnosing GISTs. The estimated incidence of GIST in the United States is approximately 5000 cases annually. This makes GIST the most common form of sarcoma, which constitutes more than 70 types of cancer. The majority of GISTs present at ages 50�70 years. Across most of the age spectrum, the incidence of GIST is similar in men and women. Adult GISTs are rare before age 40. Pediatric GISTs are considered to be biologically distinct. Unlike GISTs at other ages, pediatric GISTs are more common in girls and young women.

* Gastroschisis represents a congenital defect characterised by a defect in the anterior abdominal wall through which the abdominal contents freely protrude. There is no overlying sac or peritoneum, and the size of the defect is usually less than 4 centimetres (1.6 in). The abdominal wall defect is located at the junction of the umbilicus and normal skin, and is almost always to the right of the umbilicus. The defect occurs 5�8 weeks after conception, most likely due to a disruption of the blood supply to the developing abdominal wall. Gastroschisis as a stand-alone congenital defect, like many birth defects when isolated, exhibits multifactorial determination with a 2-3% recurrence risk for subsequent pregnancies.The morbidity is closely related to the presence of other malformations and complications of the wound or the intestine. The malformation is slightly more frequent in males than females. The frequency of gastroschisis is associated with young maternal age, low number of gestations, alcohol, tobacco and NSAID use, and infections. This abnormality was historically reported as having a ratio of 1:10,000 and is usually detected before birth. It has been reported that the incidence of gastroschisis has increased in recent years.

* General fibrosis syndrome, replacement of normal muscle tissue by fibrous tissue of varying degrees.

* General prophylaxis, disease prevention.

* Gerstmann-Straussler-Scheinker disease (GSS) is an extremely rare, neurodegenerative brain disorder. It is almost always inherited and is found in only a few families around the world. Onset of the disease usually occurs between the ages of 35 and 55. In the early stages, patients may experience varying levels of ataxia (lack of muscle coordination), including clumsiness, unsteadiness, and difficulty walking. As the disease progresses, the ataxia becomes more pronounced and most patients develop dementia. Other symptoms may include dysarthria (slurring of speech), nystagmus (involuntary movements of the eyes), spasticity (rigid muscle tone), and visual disturbances, sometimes leading to blindness. Deafness also can occur. In some families, parkinsonian features are present. GSS belongs to a family of human and animal diseases known as the transmissible spongiform encephalopathies (TSEs). Other TSEs include Creutzfeldt-Jakob disease, kuru, and fatal familial insomnia.

* Gestational diabetes also known as gestational diabetes mellitus (GDM), is when a women without diabetes, develop high blood sugar levels during pregnancy. Gestational diabetes generally results in few symptoms. It; however, increases the risk of pre-eclampsia, depression, and requiring a Caesarean section. Babies born to mothers with poorly treated gestational diabetes are at increased risk of being too large, having low blood sugar after birth, and jaundice. If untreated, it can also result in a stillbirth. Long term, children are at higher risk of being overweight and developing type 2 diabetes.

* Giant lymph node hyperplasia (also known as Castleman's disease or CD) is a rare, benign disease involving lymph nodes in different locations, predominantly in the mediastinum. This disorder is marked by non-cancerous growths (tumors) that may develop in the lymph node tissue at a single site or throughout the body. It involves hyperproliferation of certain B cells that typically produce cytokines. The most common 'B Symptoms' of of Castleman's disease are high fevers, anemia, weight loss, loss of appetite, and low white blood cell counts, which may to be due to the overproduction of interleukin 6. Symptomatically, therefore, this disease can be difficult to diagnose and even in the case of a lymph-node biopsy a conclusive diagnosis remains problematic. The exact cause of Castleman's Disease is not known, though some researchers speculate that increased production of interleukin-6 (IL-6) may be involved in the development of Castleman's Disease.

* Giardiasis (popularly known as beaver fever) is a zoonotic parasitic disease caused by the flagellate protozoan Giardia lamblia (also sometimes called Giardia intestinalis and Giardia duodenalis). The giardia organism inhabits the digestive tract of a wide variety of domestic and wild animal species, as well as humans. It is one of the most common pathogenic parasitic infections in humans worldwide. Symptoms vary from none to severe diarrhea with poor absorption of nutrients. It can result in weakness, loss of appetite, stomach cramps, vomiting, bloating, excessive gas, and burping. Symptoms typically develop 9�15 days after exposure, but may occur as early as one day. Symptoms are caused by Giardia organisms infecting the cells of the duodenum and jejunum of the small intestine and blocking nutrient absorption. Most people are asymptomatic; only about a third of those infected exhibit symptoms. If the infection is not treated, these symptoms may last for six weeks or more.

* Gilbert disease is a relatively mild condition characterized by periods of elevated levels of a toxic substance called bilirubin in the blood (hyperbilirubinemia). Bilirubin, which has an orange-yellow tint, is produced when red blood cells are broken down. This substance is removed from the body only after it undergoes a chemical reaction in the liver, which converts the toxic form of bilirubin (unconjugated bilirubin) to a nontoxic form called conjugated bilirubin. People with Gilbert disease have a buildup of unconjugated bilirubin in their blood (unconjugated hyperbilirubinemia). In affected individuals, bilirubin levels fluctuate and very rarely increase to levels that cause jaundice, which is yellowing of the skin and whites of the eyes. Gilbert disease is usually recognized in adolescence. If people with this condition have episodes of hyperbilirubinemia, these episodes are generally mild and typically occur when the body is under stress, for instance because of dehydration, prolonged periods without food (fasting), illness, vigorous exercise, or menstruation. Some people with Gilbert syndrome also experience abdominal discomfort or tiredness. However, approximately 30 percent of people with Gilbert syndrome have no signs or symptoms of the condition.

* Gingivitis ("inflammation of the gum tissue") is a non-destructive periodontal disease. The most common form of gingivitis, and the most common form of periodontal disease overall, is in response to bacterial biofilms (also called plaque) adherent to tooth surfaces, termed plaque-induced gingivitis.

* Glaucoma, disease in which damage to the optic nerve leads to progressive, irreversible vision loss.

* Glioblastoma, also known as glioblastoma multiforme (GBM), is the most common and most aggressive cancer that begins within the brain. Signs and symptoms are initially non-specific. They may include headaches, personality changes, nausea, and symptoms similar to those of a stroke. Worsening of symptoms is often rapid. This can progress to unconsciousness. Although common symptoms of the disease include seizure, nausea and vomiting, headache, memory loss, and hemiparesis, the single most prevalent symptom is a progressive memory, personality, or neurological deficit due to temporal and frontal lobe involvement. The kind of symptoms produced depends more on the location of the tumor than on its pathological properties. The tumor can start producing symptoms quickly, but occasionally is an asymptomatic condition until it reaches an enormous size. For unknown reasons, GBM occurs more commonly in males. Most glioblastoma tumors appear to be sporadic, without any genetic predisposition.

* Glioma is a type of tumor that starts in the brain or spine. It is called a glioma because it arises from glial cells. The most common site of gliomas is the brain. Gliomas make up about 30% of all brain and central nervous system tumors and 80% of all malignant brain tumors. Symptoms of gliomas depend on which part of the central nervous system is affected. A brain glioma can cause headaches, vomiting, seizures, and cranial nerve disorders as a result of increased intracranial pressure. A glioma of the optic nerve can cause visual loss. Spinal cord gliomas can cause pain, weakness, or numbness in the extremities. Gliomas do not metastasize by the bloodstream, but they can spread via the cerebrospinal fluid and cause "drop metastases" to the spinal cord.

* Glomerulonephritis (GN), also known as glomerular nephritis, is a term used to refer to several kidney diseases (usually affecting both kidneys). Many of the diseases are characterised by inflammation either of the glomeruli or of the small blood vessels in the kidneys, hence the name, but not all diseases necessarily have an inflammatory component. As it is not strictly a single disease, its presentation depends on the specific disease entity: it may present with isolated hematuria and/or proteinuria (blood or protein in the urine); or as a nephrotic syndrome, a nephritic syndrome, acute kidney injury, or chronic kidney disease. Primary causes are intrinsic to the kidney. Secondary causes are associated with certain infections (bacterial, viral or parasitic pathogens), drugs, systemic disorders (SLE, vasculitis), or diabetes. Glomerulonephritis refers to an inflammation of the glomerulus, which is the unit involved in filtration in the kidney. This inflammation typically results in one or both of the nephrotic or nephritic syndromes. The nephrotic syndrome is characterised by the finding of edema in a person with increased protein in the urine and decreased protein in the blood, with increased fat in the blood. The nephritic syndrome is characterised by blood in the urine (especially Red blood cell casts with dysmorphic red blood cells) and a decrease in the amount of urine in the presence of hypertension. Nonproliferative glomerulonephritis is characterised by forms of glomerulonephritis in which the number of cells is not changed. These forms usually result in the nephrotic syndrome. Proliferative glomerulonephritis is characterised by an increased number of cells in the glomerulus. These forms usually present with a triad of blood in the urine, decreased urine production, and hypertension, the nephritic syndrome. These forms usually progress to end-stage kidney failure (ESKF) over weeks to years (depending on type).

* Glossopharyngeal neuralgia consists of recurring attacks of severe pain in the back of the throat, the area near the tonsils, the back of the tongue, part of the ear, and/or the area under the back of the jaw. The pain is due to malfunction of the 9th cranial nerve (glossopharyngeal nerve) The glossopharyngeal nerve helps move the muscles of the throat and carries information from the throat, tonsils, and tongue to the brain. In glossopharyngeal neuralgia, attacks are brief and occur intermittently, but they cause excruciating pain. Attacks may be triggered by a particular action, such as chewing, swallowing, talking, yawning, coughing, or sneezing. The pain usually begins at the back of the tongue or back of the throat. Sometimes pain spreads to the ear or the area at the back of the jaw. The pain may last several seconds to a few minutes and usually affects only one side of the throat and tongue. The cause is often unknown but sometimes is an abnormally positioned artery that puts pressure on (compresses) the glossopharyngeal nerve. Glossopharyngeal neuralgia, a rare disorder, usually begins after age 40 and occurs more often in men. Often, its cause is unknown.

* Glutaric acidemia type 1 is an inherited disorder in which the body is unable to break down completely the amino acids lysine, hydroxylysine and tryptophan. Excessive levels of their intermediate breakdown products (glutaric acid, glutaryl-CoA, 3-hydroxyglutaric acid, glutaconic acid) can accumulate and cause damage to the brain (and also other organs, but particularly the basal ganglia, which are regions that help regulate movement. GA1 causes secondary carnitine deficiency, as glutaric acid, like other organic acids, is detoxified by carnitine. Mental retardation may also occur. The severity of glutaric acidemia type 1 varies widely; some individuals are only mildly affected, while others have severe problems. GA1 can be defined as two clinical entities: GA1 before the encephalopathic crisis and GA1 after the encephalopathic crisis. Before the encephalopathic crisis, babies with glutaric acidemia type 1 often are born with unusually large heads (macrocephaly). After the encephalopatic crisis, affected individuals may have difficulty moving and may experience spasms, jerking, rigidity or decreased muscle tone and muscle weakness (which may be the result of secondary carnitine deficiency). Some individuals with glutaric acidemia have developed bleeding in the brain or eyes that could be mistaken for the effects of child abuse. Glutaric aciduria type 1, in many cases, can be defined as a cerebral palsy of genetic origins.

* Glycogenosis or Glycogen storage disorder, are a group of inherited diseases. They result from a problem with one of the proteins (known as enzymes) involved in the conversion of glucose to glycogen, or the breakdown of glycogen back into glucose. They mostly tend to affect the liver and muscles. Most are diagnosed in childhood. Symptoms include weakness, tiredness and low blood sugar levels. Because there are a number of different enzymes involved in glycogen production and breakdown, there are a number of different glycogenosis. Glycogen storage disorders are rare. It is estimated that about one baby in every 20,000 to 40,000 babies born has a glycogen storage disorder.

* Glycoprotein Syndrome is one of several rare inborn errors of metabolism in which glycosylation of a variety of tissue proteins and/or lipids is deficient or defective. Congenital disorders of glycosylation are sometimes known as CDG syndromes. They often cause serious, sometimes fatal, malfunction of several different organ systems (especially the nervous system, muscles, and intestines) in affected infants. The specific problems produced differ according to the particular abnormal synthesis involved. Common manifestations include ataxia; seizures; retinopathy; liver fibrosis; coagulopathies; failure to thrive; dysmorphic features (e.g., inverted nipples and subcutaneous fat pads; and strabismus.

* Gonadal disorders, related to both sexes reproductory organs.

* Gonorrhea, also spelled gonorrhoea, is a sexually transmitted infection caused by the bacterium Neisseria gonorrhoeae. Many people have no symptoms. Men may have burning with urination, discharge from the penis, or testicular pain. Women may have burning with urination, vaginal discharge, vaginal bleeding between periods, or pelvic pain. Complications in women include pelvic inflammatory disease and in men include inflammation of the epididymis. If untreated gonorrhea can occasionally spread to affect joints or heart valves. Gonorrhea is spread through sexual contact with an infected person. This includes oral, anal, and vaginal sex. It can also spread from a mother to a child during birth. Half of women with gonorrhea do not have symptoms, whereas others have vaginal discharge, lower abdominal pain, or pain with sexual intercourse associated with inflammation of the uterine cervix. Most infected men with symptoms have inflammation of the penile urethra associated with a burning sensation during urination and discharge from the penis. In men, discharge with or without burning occurs in half of all cases and is the most common symptom of the infection.

* Goodpasture syndrome is a rare and potentially life-threatening autoimmune disease. It occurs when the immune system mistakenly attacks the basement membrane in lungs and kidneys, leading to bleeding from the lungs and kidney failure. The antiglomerular basement membrane (GBM) antibodies primarily attack the kidneys and lungs, although, generalized symptoms like malaise, weight loss, fatigue, fever, and chills are also common, as are joint aches and pains. 60 to 80% of those with the condition experience both lung and kidney involvement; 20-40% have kidney involvement alone, and less than 10% have lung involvement alone. Lung symptoms usually antedate kidney symptoms and usually include: coughing up blood, chest pain (in less than 50% of cases overall), cough, and shortness of breath. Kidney symptoms usually include blood in the urine, protein in the urine, unexplained swelling of limbs or face, high amounts of urea in the blood, and high blood pressure.

* Granuloma annulare is a benign, asymptomatic, self-limited papular eruption found in patients of all ages. The primary skin lesion usually is grouped papules in an enlarging annular shape, with color ranging from flesh-colored to erythematous. These lesions can be very unsightly, but normally cause no additional symptoms and don�t have any lasting influence on an individual�s health. But, granuloma annulare is hard to deal with if the lesions affect the appearance. The two most common types of granuloma annulare are localized, which typically is found on the lateral or dorsal surfaces of the hands and feet; and disseminated, which is widespread. Localized disease generally is self-limited and resolves within one to two years, whereas disseminated disease lasts longer with some having a chronic course lasting up to 10 years.

* Granuloma is an inflammation found in many diseases. It is a collection of immune cells known as histiocytes (macrophages). Granulomas form when the immune system attempts to wall off substances it perceives as foreign but is unable to eliminate. Such substances include infectious organisms including bacteria and fungi, as well as other materials such as keratin and suture fragments.

* Granuloma pseudopyogenic usually presents with pink to red-brown, dome-shaped, solitary or multiple benign cutaneous nodules comprised of immature and mature vascular structures intermingled with endothelial cells and a varied infiltrate of eosinophils, histiocytes, lymphocytes, and mast cells. It has been suggested as a feature of IgG4-related skin disease.

* Granuloma venereum (also known as donovanosis) is a bacterial disease caused by Klebsiella granulomatis characterized by ulcerative genital lesions. It is endemic in many less developed regions. Small, painless nodules appear after about 10�40 days of the contact with the bacteria. Later, the nodules burst, creating open, fleshy, oozing lesions. The infection spreads, mutilating the infected tissue. The infection will continue to destroy the tissue until treated. The lesions occur at the region of contact typically found on the shaft of the penis, the labia, or the perineum. Rarely, the vaginal wall or cervix is the site of the lesion. The destructive nature of donovanosis also increases the risk of superinfection by other pathogenic microbes.

* Guinea worm disease is caused by drinking water contaminated by water fleas (microscopic arthropods known as copepods) that host the Dracunculus larva. Guinea worm disease used to thrive in some of the world�s poorest areas, particularly those with limited or no access to clean water. Once inside the body, stomach acid digests the water flea, but not the guinea worm larvae that are sheltered inside. These larvae find their way to the body cavity where the female mates with a male guinea worm. This takes place approximately three months after infection. The male worm dies after mating and is absorbed. The female, which contains larvae, burrows into the deeper connective tissues or adjacent to long bones or joints of the extremities. Approximately one year after the infection began, the worm creates a blister in the human host�s skin�usually on the leg or foot. Within 72 hours the blister ruptures, exposing one end of the emergent worm. This blister causes a very painful burning sensation as the worm emerges. Infected persons often immerse the affected limb in water to relieve the burning sensation. Once the blister or open sore is submerged in water, the adult female releases hundreds of thousands of guinea worm larvae, contaminating the water supply.

* Gynecomastia, abnormal development of large mammary glands in males resulting in breast enlargement.

* Gyrate atrophy of the choroid and retina is an inborn error of ornithine metabolism, caused by decreased activity of the enzyme ornithine aminotransferase. Clinically, it presents initially with poor night vision, which slowly progresses to total blindness. It is believed to be inherited in an autosomal recessive manner. Quite often, the presenting symptom of ornithine aminotransferase (OAT) deficiency is myopia which progresses to night blindness. The onset of myopia is often in early childhood. Ophthalmological findings in affected individuals include constricted visual fields, posterior subcapsular cataracts (can begin in late teens), elevated dark adaptation thresholds and decreased or absent electroretinographic responses. Symptoms of OAT deficiency are progressive, and between the ages of 45 and 65, most affected individuals are almost completely blind. In some cases, affected individuals will present in the neonatal period with disease that closely mimics a classic urea cycle defect, such as ornithine transcarbamylase deficiency, as the block in ornithine metabolism leads to secondary dysfunction of the urea cycle. These individuals present with hyperammonemia, poor feeding, failure to thrive and increased excretion of orotic acid.