The most modern format of medicine of the Digital World
Treatment combo Sessions of Modern Medicine in Digital format - N
The frequencies used in these sessions are based upon Rife sets for resonant therapy devices masked in Algorithmic piano music.
More information regarding the items in the list is given below the list.
List of Treatment combo Sessions of Modern Medicine in Digital format for problems/products available at us for just Rs. 1,000/- for any 5 sessions from any one or multiple Treatment combo Sessions for 30 doses per session (2 times a day for 15 days) in max 15 days.
1) Nail Disease
2) Nail Fungus
3) Nasal Polyps
4) Nausea
5) Neck Pain
6) Nephritis Familial
7) Nephroblastoma
8) Nephrosis
9) Nephrotic Syndrome
10) Nerve Compression Syndromes
11) Neural Tube Defects
12) Neuralgia
13) Neuralgic Amyotrophy
14) Neuroendocrine Tumors
15) Neurofibromatosis
16) Neuroleptic Malignant Syndrome
17) Neurologic Disorders
18) Neuroma Acoustic
19) Neuromuscular Diseases
20) Neuromyelitis Optica
21) Neuromyotonia
22) Neuropathy Hereditary Motor-Sensory Type
23) Neuropathy Hereditary-Autonomic Type
24) Neutropenia
25) Nevus
26) Nevus Flammeus
27) Niemann-Pick Disease
28) Nocardia Infections
29) Nocturia
30) Nocturnal Enuresis
31) Non-Hodgkin Lymphoma
32) Normal Pressure Hydrocephalus
33) Nose Congestion
34) Nose Diseases
35) Nystagmus
* Nail diseases are distinct from diseases of the skin. Although nails are a
skin appendage, they have their own signs and symptoms which may relate to other
medical conditions. Deformity or disease of the nails may be referred to as
onychosis.
* Nail fungus or Onychomycosis is the most common disease of the nails and
constitutes about half of all nail abnormalities. This condition may affect
toenails or fingernails, but toenail infections are particularly common. It
occurs in about 10 percent of the adult population.
* Nephritis familial or Alport syndrome is a genetic disorder affecting around 1
in 50,000 children, characterized by glomerulonephritis, end-stage kidney
disease, and hearing loss. Alport syndrome can also affect the eyes, though the
changes do not usually affect sight, except when changes to the lens occur in
later life. Blood in urine is universal. Proteinuria is a feature as kidney
disease progresses.
* Nephroblastoma, also called Wilms’ tumor, is a rare type of kidney cancer that
primarily affects children. Although rare, Wilms’ tumor is the most common type
of kidney cancer in children, and the fourth most common type of childhood
cancer overall. In Wilms tumor, one or more tumors may be found in one or both
kidneys. Wilms tumor may spread to the lungs, liver, bone, brain, or nearby
lymph nodes. Children with Wilms’ tumor might display the following symptoms:
constipation, abdominal pain, swelling, or discomfort, a lump in the abdomen,
nausea and vomiting, weakness and fatigue, loss of appetite, a fever for no
reason, blood in their urine or discoloration of their urine, high blood
pressure, which may cause chest pain, shortness of breath, and headaches,
increased and uneven growth of one side of their body. Many children with Wilms’
tumor also have birth defects.
* Nephrosis is any of various forms of kidney disease (nephropathy). In an old
and broad sense of the term, it is any nephropathy, but in current usage the
term is usually restricted to a narrower sense of nephropathy without
inflammation or neoplasia, in which sense it is distinguished from nephritis,
which involves inflammation. It is also defined as any purely degenerative
disease of the renal tubules. Nephrosis is characterized by a set of signs
called the nephrotic syndrome. Nephrosis can be a primary disorder or can be
secondary to another disorder. Nephrotic complications of another disorder can
coexist with nephritic complications. In other words, nephrosis and nephritis
can be pathophysiologically contradistinguished, but that does not mean that
they cannot occur simultaneously.
* Nephrotic syndrome is a syndrome comprising signs of nephrosis, chiefly
proteinuria, hypoalbuminemia, and edema. It is a component of
glomerulonephrosis, in which different degrees of proteinuria occur.
Essentially, loss of protein through the kidneys (proteinuria) leads to low
protein levels in the blood (hypoproteinemia including hypoalbuminemia), which
causes water to be drawn into soft tissues (edema). Very low hypoalbuminemia can
also cause a variety of secondary problems, such as water in the abdominal
cavity (ascites), around the heart or lung (pericardial effusion, pleural
effusion), high cholesterol (hence hyperlipidemia), loss of molecules regulating
coagulation (hence increased risk of thrombosis).
* Neural tube defects (NTDs) are a group of conditions in which an opening in
the spinal cord or brain remains from early in human development. In the 3rd
week of pregnancy called gastrulation, specialized cells on the dorsal side of
the embryo begin to change shape and form the neural tube. When the neural tube
does not close completely, an NTD develops.
* Neuralgic amyotrophy, characterized by episodes of severe pain and muscle
wasting (amyotrophy) in one or both shoulders and arms.
* Neuroendocrine tumors (NETs) are neoplasms that arise from cells of the
endocrine (hormonal) and nervous systems. Many are benign, while some are
malignant. They most commonly occur in the intestine, where they are often
called carcinoid tumors, but they are also found in the pancreas, lung and the
rest of the body. Although there are many kinds of NETs, they are treated as a
group of tissue because the cells of these neoplasms share common features, such
as looking similar, having special secretory granules, and often producing
biogenic amines and polypeptide hormones. In the category of
Gastroenteropancreatic neuroendocrine tumors (GEP-NET), carcinoids most commonly
affect the small bowel, particularly the ileum, and are the most common
malignancy of the appendix. Many tumors do not cause symptoms even when they
have metastasized. Other tumors even if very small can produce adverse effects
by secreting hormones. A carcinoid crisis with profound flushing, bronchospasm,
tachycardia, and widely and rapidly fluctuating blood pressure can occur if
large amounts of hormone are acutely secreted, which is occasionally triggered
by factors such as diet, alcohol, surgery chemo therapy, embolization therapy or
radiofrequency ablation. About one third of GEP-NETs are Pancreatic
neuroendocrine tumors (PanNETs) often referred to as "islet cell tumors", or
"pancreatic endocrine tumors". PanNETs are quite distinct from the usual form of
pancreatic cancer, adenocarcinoma, which arises in the exocrine pancreas. Well
or intermediately differentiated PanNETs are sometimes called islet cell tumors.
In addition to the two main categories of GEP-NET, there are rarer forms of
neuroendocrine tumors that arise anywhere in the body, including within the
lung, thymus and parathyroid. Bronchial carcinoid can cause airway obstruction,
pneumonia, pleurisy, difficulty with breathing, cough, and hemoptysis, or may be
associated with weakness, nausea, weight loss, night sweats, neuralgia, and
Cushing’s syndrome. Some are asymptomatic.
* Neurofibromatosis, genetic disease in which patients develop multiple soft
tumors (neurofibromas).
* Neuroleptic malignant syndrome is a life-threatening, neurological disorder
most often caused by an adverse reaction to neuroleptic or antipsychotic drugs.
Symptoms include high fever, sweating, unstable blood pressure, stupor, muscular
rigidity, and autonomic dysfunction. In most cases, the disorder develops within
the first 2 weeks of treatment with the drug; however, the disorder may develop
any time during the therapy period. The syndrome can also occur in people taking
anti-Parkinsonism drugs known as dopaminergics if those drugs are discontinued
abruptly.
* Neurologic disorder is any disorder of the body nervous system. Structural,
biochemical or electrical abnormalities in the brain, spinal cord or other
nerves can result in a range of symptoms. Examples of symptoms include
paralysis, muscle weakness, poor coordination, loss of sensation, seizures,
confusion, pain and altered levels of consciousness. There are many recognized
neurological disorders, some relatively common, but many rare. The specific
causes of neurological problems vary, but can include genetic disorders,
congenital abnormalities or disorders, infections, lifestyle or environmental
health problems including malnutrition, and brain injury, spinal cord injury or
nerve injury.
* Neuromyelitis optica (NMO) is an uncommon disease syndrome of the central
nervous system (CNS) that affects the optic nerves and spinal cord. Individuals
with NMO develop optic neuritis, which causes pain in the eye and vision loss,
and transverse myelitis, which causes weakness, numbness, and sometimes
paralysis of the arms and legs, along with sensory disturbances and loss of
bladder and bowel control. NMO leads to loss of myelin, which is a fatty
substance that surrounds nerve fibers and helps nerve signals move from cell to
cell. The syndrome can also damage nerve fibers and leave areas of broken-down
tissue. In the disease process of NMO, for reasons that aren’t yet clear, immune
system cells and antibodies attack and destroy myelin cells in the optic nerves
and the spinal cord.
* Neuromyotonia is a form of peripheral nerve hyperexcitability that causes
spontaneous muscular activity resulting from repetitive motor unit action
potentials of peripheral origin. As a result of muscular hyperactivity patients
may present with muscle cramps, stiffness, myotonia-like symptoms (slow
relaxation), associated walking difficulties, hyperhidrosis (excessive
sweating), myokymia (quivering of a muscle), fasciculations (muscle twitching),
fatigue, exercise intolerance, myoclonic jerks and other related symptoms. The
symptoms (especially the stiffness and fasciculations) are most prominent in the
calves, legs, trunk, and sometimes the face and neck, but can also affect other
body parts. NMT symptoms may fluctuate in severity and frequency. Symptoms range
from mere inconvenience to debilitating. At least a third of people also
experience sensory symptoms.
* Neuropathy hereditary autonomic type4, group of disorders characterized by the
loss of sensation (sensory loss), especially in the feet and legs and, less
severely, in the hands and forearms.
* Neuropathy hereditary motor-sensory type3, group of neuropathies affecting
motor and sensory peripheral nerves.
* Neutropenia, abnormally low number of neutrophils in the blood, which leads to
increased susceptibility to disease.
* Nevus flammeus, port-wine stain or firemark, is almost always a birthmark; in
rare cases it can develop in early childhood. It is caused by a vascular anomaly
(a capillary malformation in the skin). Port-wine stains are named for their
coloration, which is similar in color to Port-wine, a fortified red wine from
Portugal. Port-wine stains ordinarily persist throughout life. The area of skin
affected grows in proportion to general growth. Port-wine stains occur most
often on the face but can appear anywhere on the body, particularly on the neck
and upper trunk. Early stains are usually flat and pink in appearance. As the
child matures, the color may deepen to a dark red or purplish color. In
adulthood, thickening of the lesion or the development of small lumps may occur.
Port-wine stains may be part of a syndrome such as Sturge–Weber syndrome or
Klippel–Trénaunay–Weber syndrome. In the absence of successful treatment,
hypertrophy (increased tissue mass) of the stains may cause problems later in
life, such as loss of function (especially if the stain is near the eye or
mouth), bleeding, and increasing disfigurement. Lesions on or near the eyelid
can be associated with glaucoma. If the port-wine stain is on the face or other
highly visible part of the body, its presence can also cause emotional and
social problems for the affected person.
* New variant Creutzfeldt - Jakob Disease in humans is thought to be caused by
prions, which encourage conformational changes of proteins. This evolutionary
process is problematic because it promotes the refolding of native proteins into
the diseased state, or in other words - from alpha-helices into beta-pleated
sheets. When these amyloid proteins accumulate in organs and tissues, the
misfolded hydrophobic proteins are not always dissolved by proteolysis. When the
exponential increase of these diseased conformations of proteins occurs, the
cells are no longer able to function properly, and eventually die off. Once a
prion is transmitted, it begins the “self-sustaining feedback loop” which causes
the disease to progress. As the diseased proteins die off, it causes the spongy
deterioration of the brain and spinal cord, leading to an array of symptoms that
are incurable, irreversible, and eventually fatal. Patients diagnosed with nvCJD
are usually diagnosed at a young age, with the disease lasting approximately
12-14 months. Symptoms affect both the physical and mental capacity of patients
leading to rapidly progressing dementia, irritability, mood swings, muscle
jerks, loss of sight, and eventually the inability to speak along with the loss
of all motor function, resulting in death.
* Niemann-Pick disease is an inherited disease that affects lipid metabolism, or
the way fats, lipids, and cholesterol are stored in or removed from the body.
People with Niemann-Pick disease have an abnormal lipid metabolism that causes a
buildup of harmful amounts of lipids in various organs. The disease primarily
affects the: liver, spleen, brain, bone marrow. This leads to enlargement of the
spleen and neurologic problems. Niemann-Pick disease involves dysfunctional
metabolism of sphingolipids, which are fats found in cell membranes, so it is a
kind of sphingolipidosis. Sphingolipidoses, in turn, are included in the larger
family of lysosomal storage diseases. Symptoms are related to the organs in
which sphingomyelin accumulates. Enlargement of the liver and spleen
(hepatosplenomegaly) may cause reduced appetite, abdominal distension, and pain.
Enlargement of the spleen (splenomegaly) may also cause low levels of platelets
in the blood (thrombocytopenia). Accumulation of sphingomyelin in the central
nervous system (including the cerebellum) results in unsteady gait (ataxia),
slurring of speech (dysarthria), and difficulty in swallowing (dysphagia). Basal
ganglia dysfunction causes abnormal posturing of the limbs, trunk, and face
(dystonia). Upper brainstem disease results in impaired voluntary rapid eye
movements (supranuclear gaze palsy). More widespread disease involving the
cerebral cortex and subcortical structures causes gradual loss of intellectual
abilities, causing dementia and seizures. Bones also may be affected: symptoms
may include enlarged bone marrow cavities, thinned cortical bone, or a
distortion of the hip bone called coxa vara. Sleep-related disorders, such as
sleep inversion, sleepiness during the day and wakefulness at night, may occur.
Gelastic cataplexy, the sudden loss of muscle tone when the affected patient
laughs, is also seen.
* Nocardiosis is a rare infection caused by the Nocardia asteroides bacterium.
This type of bacteria can be found in the soil and water of regions around the
world. People may become infected with this bacteria when they inhale it or when
the bacteria enter an open wound. The infection can’t be spread from one person
to another. Nocardiosis most commonly occurs in the lungs, but it may spread to
other areas of the body, such as the: skin, digestive system, brain, kidneys,
heart, eyes, bones. Though nocardiosis can develop in anyone, the condition is
much more likely to affect people with very weak immune systems. The symptoms
will vary depending on the area of the body that has become infected.
Nocardiosis most commonly affects the lungs with fever, fatigue, chest pain, a
cough, night sweats. The skin is the second most commonly affected area with
open, seeping sores, ulcers, rashes, swollen lymph nodes. In some cases, the
infection can spread to other parts of the body and cause varying symptoms. In
the digestive system, nausea, vomiting, sudden weight loss, abdominal swelling.
In the brain, seizures, headaches, confusion, dizziness.
* Nocturia, to wake at night one or more times for urinating.
* Non-Hodgkin lymphoma (NHL) is a group of blood cancers that includes all types
of lymphoma except Hodgkin's lymphomas. Symptoms include enlarged lymph nodes,
fever, night sweats, weight loss, and feeling tired. Other symptoms may include
bone pain, chest pain, or itchiness. Some forms are slow growing while others
are fast growing. Lymphomas are types of cancer that develops from lymphocytes,
a type of white blood cell. Risk factors include poor immune function,
autoimmune diseases, Helicobacter pylori infection, hepatitis C, obesity, and
Epstein-Barr virus infection. The World Health Organization (WHO) classifies
lymphomas into five major groups, including one for Hodgkin's lymphoma. Within
the four groups for NHL there are over 60 specific types of lymphoma. In 2013
about 2.96 million people had non-Hodgkin lymphoma and 226,000 died. In the
United States 2.1% of people are affected at some point in their life. The most
common age of diagnosis is between 65 to 75 years old. The percentage of people
who survive five years in the United States is 71%. Symptoms include enlarged
lymph nodes, fever, night sweats, weight loss, and feeling tired. Other symptoms
may include bone pain, chest pain, or itchiness. Some forms are slow growing
while others are fast growing. Possible causes and associations with at least
some forms of NHL include the following: Infectious agents: Epstein-Barr virus:
associated with Burkitt's lymphoma, Hodgkin's lymphoma, follicular dendritic
cell sarcoma, extranodal NK-T-cell lymphoma. Human T-cell leukemia virus:
associated with adult T-cell lymphoma. Helicobacter pylori: associated with
gastric lymphoma. HHV-8: associated with primary effusion lymphoma, multicentric
Castleman disease. Hepatitis C virus: associated with splenic marginal zone
lymphoma, lymphoplasmacytic lymphoma and diffuse large B-cell lymphoma. HIV
infection. Some chemicals, like polychlorinated biphenyls (PCBs),
diphenylhydantoin, dioxin, and phenoxy herbicides. Medical treatments, like
radiation therapy and chemotherapy. Genetic diseases, like Klinefelter's
syndrome, Chédiak-Higashi syndrome, ataxia telangiectasia syndrome. Autoimmune
diseases, like Sjögren’s syndrome, celiac disease, rheumatoid arthritis, and
systemic lupus erythematosus. NHL is the sixth most common cancer in the UK
(around 12,800 people were diagnosed with the disease in 2011), and it is the
eleventh most common cause of cancer death (around 4,700 people died in 2012).
* Non-Malignant (Benign) Tumor is a mass of cells (tumor) that lacks the ability
to invade neighboring tissue or metastasize. These characteristics are required
for a tumor to be defined as cancerous and therefore benign tumors are
non-cancerous. Also, benign tumors generally have a slower growth rate than
malignant tumors and the tumor cells are usually more differentiated (cells have
normal features). Benign tumors are typically surrounded by an outer surface
(fibrous sheath of connective tissue) or remain with the epithelium. Common
examples of benign tumors include moles and uterine fibroids.
* Non-small-cell lung carcinoma (NSCLC) is any type of epithelial lung cancer
other than small cell lung carcinoma (SCLC). NSCLC accounts for about 85% of all
lung cancers. The most common types of NSCLC are squamous cell carcinoma, large
cell carcinoma, and adenocarcinoma, but there are several other types that occur
less frequently, and all types can occur in unusual histologic variants and as
mixed cell-type combinations. Adenocarcinoma of the lung is currently the most
common type of lung cancer in "never smokers" (lifelong non-smokers).
Adenocarcinomas account for approximately 40% of lung cancers. Squamous cell
carcinoma (SCC) of the lung is more common in men than in women. It is closely
correlated with a history of tobacco smoking, more so than most other types of
lung cancer. Large cell lung carcinoma (LCLC) is a heterogeneous group of
undifferentiated malignant neoplasms originating from transformed epithelial
cells in the lung. LCLC's have typically comprised around 10% of all NSCLC in
the past, although newer diagnostic techniques seem to be reducing the incidence
of diagnosis of "classic" LCLC in favor of more poorly differentiated squamous
cell carcinomas and adenocarcinomas. LCLC is, in effect, a "diagnosis of
exclusion", in that the tumor cells lack light microscopic characteristics that
would classify the neoplasm as a small-cell carcinoma, squamous-cell carcinoma,
adenocarcinoma, or other more specific histologic type of lung cancer. LCLC is
differentiated from small cell lung carcinoma (SCLC) primarily by the larger
size of the anaplastic cells, a higher cytoplasmic-to-nuclear size ratio, and a
lack of "salt-and-pepper" chromatin.
* Normal pressure hydrocephalus, clinical symptom complex caused by the build-up
of cerebrospinal fluid.
* Nystagmus, involuntary eye movement.