Treatment combo Sessions of Modern Medicine in Digital format

Treatment combo Sessions of Modern Medicine in Digital format - R

The frequencies used in these sessions are based upon Rife sets for resonant therapy devices masked in Algorithmic piano music.

More information regarding the items in the list is given below the list.

List of Treatment combo Sessions of Modern Medicine in Digital format for problems/products available at us for just Rs. 1,000/- for any 5 sessions from any one or multiple Treatment combo Sessions for 30 doses per session (2 times a day for 15 days) in max 15 days.

1) Rachitis
2) Rat-Bite Fever
3) Raynaud Disease
4) Rectal Diseases
5) Rectal Prolapse
6) Reflex Sympathetic Dystrophy
7) Refractive Errors
8) Relapsing Fever
9) Renal Artery Stenosis
10) Renal Osteodystrophy
11) Respiratory Distress Syndrome-Newborn
12) Respiratory Tract Infections
13) Retinal Detachment
14) Retinal Diseases
15) Retinitis Pigmentosa
16) Retinoblastoma
17) Retinopathy of Prematurity
18) Retinoschisis
19) Retroperitoneal Fibrosis
20) Retropharyngeal Abscess
21) Rett Syndrome
22) Rhabdomyolysis
23) Rheumatic Disease
24) Rheumatic Fever
25) Rhinitis
26) Rickettsia Infections
27) Right Ventricular Hypertrophy
28) Romberg Disease
29) Rosacea
30) Roseola Infantum
31) Rotor Syndrome
32) Rubella
33) Rubinstein-Taybi Syndrome

* Rachitis, also rickets, is a skeletal disorder that�s caused by a lack of vitamin D, calcium, or phosphate. These nutrients are important for the development of strong, healthy bones. People with rickets may have weak and soft bones, stunted growth, and, in severe cases, skeletal deformities. Rickets is most common in children who are between 6 and 36 months old. Children are at the highest risk of rickets because they�re still growing. Children might not get enough vitamin D if they live in a region with little sunlight, follow a vegetarian diet, or don�t drink milk products. In some cases, the condition is hereditary. Symptoms of rickets include: Pain or tenderness in the bones of the arms, legs, pelvis, or spine. Stunted growth and short stature. Bone fractures. Muscle cramps. Teeth deformities. Skeletal deformities.

* Rat-bite fever is an acute, febrile human illness caused by bacteria transmitted by rodents, rats, or mice, in most cases, which is passed from rodent to human by the rodent's urine or mucous secretions. It is a rare disease spread by infected rodents and can be caused by two specific types of bacteria. Some cases are diagnosed after patients were exposed to the urine or bodily secretions of an infected animal. These secretions can come from the mouth, nose, or eyes of the rodent. The majority of cases are due to the animal's bite. It can also be transmitted throughout food or water contaminated with rat feces or urine. Household pets such as dogs or cats exposed to these animals can also carry the disease and infect humans. Symptoms are different for every person depending on the type of rat-bite fever with which the person is infected. Both types of rat-bite fever, spirillary and streptobacillary, have a few individual symptoms, although most symptoms are shared. Rat-bite symptoms are visually seen in most cases and include inflammation around the open sore. A rash can also spread around the area and appear red or purple. Other symptoms associated with streptobacillary rat-bite fever include chills, fever, vomiting, headaches, and muscle aches. Joints can also become painfully swollen and pain can be experienced in the back. Skin irritations such as ulcers or inflammation can develop on the hands and feet. Wounds heal slowly, so symptoms possibly come and go over the course of a few months. Symptoms associated with spirillary rat-bite fever include issues with the lymph nodes, which often swell or become inflamed as a reaction to the infection. The most common locations of lymph node swelling are in the neck, groin, and underarm. Symptoms generally appear within 2 to 10 days of exposure to the infected animal. It begins with the fever and progresses to the rash on the hands and feet within 2 to 4 days. Rash appears all over the body with this form, but rarely causes joint pain.

* Raynaud's disease is excessively reduced blood flow in response to cold or emotional stress, causing discoloration of the fingers, toes, and occasionally other areas. This condition may also cause nails to become brittle with longitudinal ridges. The phenomenon is believed to be the result of vasospasms that decrease blood supply to the respective regions. Its pathophysiology includes hyperactivation of the sympathetic nervous system causing extreme vasoconstriction of the peripheral blood vessels, leading to tissue hypoxia. Chronic, recurrent cases of Raynaud's phenomenon can result in atrophy of the skin, subcutaneous tissues, and muscle. In rare cases it can cause ulceration and ischemic gangrene.

* Rectal prolapse is protrusion of rectal tissue through the anus to the exterior of the body. The rectum is the final section of the large intestine. Rectal prolapse can be either partial or complete. In partial prolapse, only the mucosa layer (mucous membrane) of the rectum extends outside the body. The projection is generally 0.75-1.5 in (2-4 cm) long. In complete prolapse, called procidentia, the full thickness of the rectum protrudes for up to 4.5 in (12 cm). Rectal prolapse is most common in people over age 60, and occurs much more frequently in women than in men. It is also more common in psychiatric patients. Prolapse can occur in normal infants, where it is usually transient. In children it is often an early sign of cystic fibrosis or is due to neurological or anatomical abnormalities. Although rectal prolapse in adults may initially reduce spontaneously after bowel movements, it eventually becomes permanent. Adults who have had prior rectal or vaginal surgery, who have chronic constipation, regularly depend on laxatives, have multiple sclerosis or other neurologic diseases, stroke, or paralysis are more likely to experience rectal prolapse.

* Refractive errors, also known as refraction errors, are a problem with focusing of light on the retina due to the shape of the eye. The most common types of refractive error are near-sightedness, far-sightedness, astigmatism, and presbyopia. Near-sightedness results in far objects being blurry, far-sightedness result in close objects being blurry, astigmatism causes objects to appear stretched out or blurry, and presbyopia results in a poor ability to focus on close objects. Other symptoms may include double vision, headaches, and eye strain.

* Relapsing fever is an infection caused by several species of bacteria in the Borrelia family. There are two major forms of relapsing fever: Tick-borne relapsing fever (TBRF) or endemic relapsing fever is transmitted by the Ornithodoros tick. The illness occurs mainly in rural communities where people are in close proximity to small mammals that carry the soft-bodied ticks, in a chronic endemic pattern. Louse-borne relapsing fever (LBRF) or epidemic relapsing fever is transmitted by body lice. Causes epidemic outbreaks of illness when public-health maintenance breaks down, eg during wars, famine, conditions of widespread poverty, displacement and overcrowding (estimated 10 million people affected during/after World War II). Sudden fever occurs within 2 weeks of infection. In LBRF, the fever usually lasts 3-6 days and is usually followed by a single, milder episode. In TRBF, multiple episodes of fever occur and each may last up to 3 days. Individuals may be free of fever for up to 2 weeks before it returns. In both forms, the fever episode may end in "crisis." This consists of shaking chills, followed by intense sweating, falling body temperature, and low blood pressure. This stage may result in death in up to 10% of people. After several cycles of fever, some people can develop dramatic central nervous system signs such as seizures, stupor, and coma. The Borrelia organism can also invade heart and liver tissues, causing inflammation of the heart muscle (myocarditis) and liver (hepatitis). Bleeding throughout the body and pneumonia are other complications.

* Renal artery stenosis is the narrowing of one or both of the renal arteries, most often caused by atherosclerosis or fibromuscular dysplasia. This narrowing of the renal artery can impede blood flow to the target kidney, resulting in renovascular hypertension � a secondary type of high blood pressure. Possible complications of renal artery stenosis are chronic kidney disease and coronary artery disease. The pathophysiology of renal artery stenosis, leads to changes in the structure of the kidney that are most noticeable in the tubular tissue. If the stenosis is longstanding and severe the glomerular filtration rate in the affected kidneys never recovers and (prerenal) kidney failure is the result. Most cases of renal artery stenosis are asymptomatic, and the main problem is high blood pressure that cannot be controlled with medication. Decreased kidney function may develop if both kidneys do not receive adequate blood flow, furthermore some people with renal artery stenosis present with episodes of flash pulmonary edema.

* Renal osteodystrophy is a bone disease that occurs when the kidneys fail to maintain proper levels of calcium and phosphorus in the blood. It's common in people with kidney disease and affects most dialysis patients. Renal osteodystrophy is most serious in children because the condition slows bone growth and causes deformities and short stature. Symptoms of renal osteodystrophy aren't usually seen in adults until they have been on dialysis for several years. If left untreated, bones thin and weaken, and symptoms include bone and joint pain, and an increased risk of fractures. In a patient with kidney failure, the kidneys stop making calcitriol, a form of vitamin D. The body then can't absorb calcium from food and starts removing it from the bones. The bone changes from renal osteodystrophy can begin many years before symptoms appear in adults with kidney disease. For this reason, it's called the "silent crippler." The symptoms of renal osteodystrophy aren't usually seen in adults until they have been on dialysis for several years. Older patients and women who have gone through menopause are at greater risk for this disease because they're already vulnerable to osteoporosis, another bone disease, even without kidney disease.

* Restrictive cardiomyopathy (RCM) is a form of cardiomyopathy in which the walls are rigid, and the heart is restricted from stretching and filling with blood properly. It is the least common of the three subtypes (JF Goodwin) of cardiomyopathy, which includes hypertrophic and dilated as well as restrictive. Rhythmicity and contractility of the heart may be normal, but the stiff walls of the heart chambers (atria and ventricles) keep them from adequately filling, reducing preload and end-diastolic volume. Thus, blood flow is reduced, and blood volume that would normally enter the heart is backed up in the circulatory system. In time, restrictive cardiomyopathy patients develop diastolic dysfunction and eventually heart failure. Untreated hearts with RCM often develop the following characteristics: Biatrial enlargement, thickened left ventricular walls (with normal chamber size), thickened right ventricular free wall (with normal chamber size), elevated right atrial pressure (>12mmHg), moderate pulmonary hypertension, normal systolic function, poor diastolic function, typically Grade III - IV Diastolic heart failure.

* Retinal detachment, separation of the light-sensitive membrane in the back of the eye (the retina) from its supporting layers. Common cause of blurred vision.

* Retinitis pigmentosa, progressive loss of photoreceptor cells leading to blindness.

* Retinoblastoma is a disease in which malignant (cancer) cells form in the tissues of the retina. The retina is the nerve tissue that lines the inside of the back of the eye. The retina senses light and sends images to the brain by way of the optic nerve. Although retinoblastoma may occur at any age, it occurs most often in children younger than 2 years. The cancer may be in one eye (unilateral) or in both eyes (bilateral). Retinoblastoma rarely spreads from the eye to nearby tissue or other parts of the body. Signs and symptoms of retinoblastoma include "white pupil" and eye pain or redness. These and other signs and symptoms may be caused by retinoblastoma or by other conditions: Pupil of the eye appears white instead of red when light shines into it. This may be seen in flash photographs of the child. Eyes appear to be looking in different directions (lazy eye). Pain or redness in the eye. Infection around the eye. Eyeball is larger than normal. Colored part of the eye and pupil look cloudy.

* Retinopathy of prematurity (ROP) is a disease of the eye affecting prematurely-born babies generally having received intensive neonatal care, in which oxygen therapy is used on them due to the premature development of their lungs. It is thought to be caused by disorganized growth of retinal blood vessels which may result in scarring and retinal detachment. ROP can be mild and may resolve spontaneously, but it may lead to blindness in serious cases. As such, all preterm babies are at risk for ROP, and very low birth weight is an additional risk factor. Both oxygen toxicity and relative hypoxia can contribute to the development of ROP.

* Retinoschisis is a condition in which an area of the retina (the tissue lining the inside of the back of the eye that transmits visual signals to the optic nerve and brain) has separated into two layers. The part of the retina that is affected by retinoschisis will have suboptimal vision. This can occur in different layers of the retina, and for different reasons. The two major causes of retinoschisis are: Juvenile X-linked Retinoschisis, a genetic disease of the retina and affects primarily boys and young men. This condition typically presents during childhood and is estimated to affect one in 5,000 to 25,000 individuals. Degenerative retinoschisis is the splitting of the retina as a result of aging. It can affect men and women. Symptos are: Decreased central vision, Decreased peripheral vision.

* Retroperitoneal fibrosis or Ormond's disease is a disease featuring the proliferation of fibrous tissue in the retroperitoneum, the compartment of the body containing the kidneys, aorta, renal tract, and various other structures. It may present with lower back pain, kidney failure, hypertension, deep vein thrombosis, and other obstructive symptoms.

* Retropharyngeal abscess is a collection of pus in the lymph nodes at the back of the throat, caused by a bacterial infection. Symptoms include difficulty and pain when swallowing, a fever, stiff neck, and noisy breathing. Because the lymph nodes at the back of the throat begin to disappear by age 4 to 5 years, retropharyngeal abscesses are more common among children aged 1 to 8 than adults. The main symptoms are difficulty and pain when swallowing, a fever, and enlargement of the lymph nodes in the neck. The voice is muffled, and children may drool. The neck may be stiff, and children may hold their head at an angle. The abscess can block the airway, making breathing difficult and noisy, particularly when children inhale (called stridor). Children may lie on their back, tilt their head and neck back, and raise their chin to make breathing easier. Adults may have severe neck pain but do not always have stridor. Complications include bleeding around the abscess, rupture of the abscess into the airway (which can block the airway), and pneumonia. The voice box (larynx) may go into spasm and further interfere with breathing. Blood clots may form in the jugular veins of the neck. Infection may spread down into the chest. Sometimes widespread inflammation and infection of the bloodstream occurs, causing organs to malfunction (a condition called septic shock).

* Rett syndrome is a rare genetic postnatal neurological disorder of the grey matter of the brain that almost exclusively affects females but has also been found in male patients. The clinical features include small hands and feet and a deceleration of the rate of head growth (including microcephaly in some). Repetitive stereotyped hand movements, such as wringing and/or repeatedly putting hands into the mouth, are also noted. People with Rett syndrome are prone to gastrointestinal disorders and up to 80% have seizures. They typically have no verbal skills, and about 50% of affected individuals do not walk. Scoliosis, growth failure, and constipation are very common and can be problematic. The signs of this disorder are most easily confused with those of Angelman syndrome, cerebral palsy and autism. Rett syndrome occurs in approximately 1:10,000 live female births in all geographies, and across all ethnicities. Initial development is normal. Onset occurs between 6 and 18 months of age. During this time there are subtle developmental deviations and early indicators of Rett syndrome. A period of developmental stagnation is followed by developmental regression where language and motor milestones regress, purposeful hand use is lost, and acquired deceleration in the rate of head growth (resulting in microcephaly in some) is seen. Hand stereotypes are typical, and breathing irregularities such as hyperventilation, breathholding, or sighing are seen in many. Early on, autistic-like behavior may be seen. The infant with Rett syndrome often avoids detection until 6�18 months, owing to a relatively normal appearance and some developmental progress. However, closer scrutiny reveals disturbance of the normal spontaneous limb and body movements that are thought to be regulated in the brainstem. The brief period of developmental progress is followed by stagnation and regression of previously acquired skills. During regression, some features are similar to those of autism. Signs may stabilize for many decades, particularly for interaction and cognitive function such as making choices. Asocial behavior may change to highly social behavior. Motor functions may slow as rigidity and dystonia appear. Seizures may be problematic, with a wide range of severity. Scoliosis occurs in most, and may require corrective surgery. Those who remain ambulatory tend to have less progression of scoliosis.

* Rhabdomyolysis is the breakdown of damaged skeletal muscle. Muscle breakdown causes the release of myoglobin into the bloodstream. Myoglobin is the protein that stores oxygen in the muscles. If there is too much myoglobin in the blood, it can cause kidney damage. Rhabdomyolysis is always triggered by muscle injury. This injury can have physical, chemical or genetic causes. Anything that damages the muscles can cause this condition. Some people develop rhabdomyolysis because of genetic conditions such as metabolic problems. Many types of infection and inflammation can cause rhabdomyolysis. The condition can also occur due to exposure to some drugs. The initial symptoms of rhabdomyolysis can be subtle. They�re not specific and may mimic other conditions. The symptoms of rhabdomyolysis include: muscle weakness, low urine output, fatigue, soreness, bruising, dark, tea-colored urine, infrequent urination, a fever, a sense of malaise, or feeling sick, nausea, vomiting, confusion, agitation.

* Rheumatic fever is an inflammatory disease that can involve the heart, joints, skin, and brain. The disease typically develops two to four weeks after a streptococcal throat infection. Signs and symptoms include fever, multiple painful joints, involuntary muscle movements, and occasionally a characteristic non-itchy rash known as erythema marginatum. The heart is involved in about half of cases. Damage to the heart valves, known as rheumatic heart disease, usually occurs after multiple attacks but can sometimes occur after a single attack. The damaged valves may result in heart failure, atrial fibrillation and infection of the valves. Rheumatic fever may occur following an infection of the throat by the bacteria Streptococcus pyogenes. If the infection is untreated rheumatic fever can occur in up to three percent of people. The underlying mechanism is believed to involve the production of antibodies against a person's own tissues. Some people due to their genetics are more likely to get the disease when exposed to the bacteria than others. Other risk factors include malnutrition and poverty.

* Right ventricular hypertrophy (RVH) is a form of ventricular hypertrophy affecting the right ventricle. Blood travels through the right ventricle to the lungs via the pulmonary arteries. If conditions occur which decrease pulmonary circulation, meaning blood does not flow well from the heart to the lungs, extra stress can be placed on the right ventricle. This can lead to right ventricular hypertrophy. It can affect ECG findings. An ECG with right ventricular hypertrophy may or may not show a right axis deviation on the graph. Common causes include: Pulmonary hypertension, Tetralogy of Fallot, pulmonary valve stenosis, pulmonic regurgitation, ventricular septal defect (VSD), high altitude, cardiac fibrosis, chronic obstructive pulmonary disease (COPD), athletic heart syndrome.

* Romberg Diseasee, also known as Progressive hemifacial atrophy, is an uncommon degenerative and poorly understood condition. It is characterized by a slow and progressive atrophy affecting one side of the face. The incidence and the cause of this alteration are unknown. A cerebral disturbance of fat metabolism has been proposed as a primary cause. This can be result of a trophic malformation of cerebral sympathetic nervous system. Possible factors that are involved in the pathogenesis are trauma, viral infections, heredity, endocrine disturbances, and autoimmunity, among others. Characteristically, atrophy progresses slowly for several years and, soon after, it become stable.

* Rosacea, chronic condition characterized by facial redness and sometimes pimples.

* Roseola infantum is a disease caused by human herpes virus type 6B (HHV-6B) and possibly type 7 (HHV-7). Roseola is characterised by high fever lasting for 3�5 days, runny nose, irritability and tiredness. As the fever subsides a rash (exanthem) may appear on the face and body. Roseola is most commonly seen in children between 6 months and 3 years of age. Most children (86%) will have had roseola by the age of 1 year. Roseola is rarely seen in adults, as infection during childhood probably confers lifelong immunity to the disease. Repeat attacks have been known to occur but are not common. Roseola is spread from person to person via saliva of asymptomatic family members. The incubation period for roseola is approximately 9�10 days after exposure. In many cases of roseola, the child appears well with few or no signs or symptoms. Typical cases are characterised by the following: High fever (often up to 40 C) for 3�5 days. Upper respiratory symptoms such as sore throat, cough, runny nose or congestion. Irritability and tiredness. Rash appears around days 3 to 5, as fever subsides. In some cases, a child may be infected with the virus and never develop the rash. Less commonly, the rash may appear without a preceding fever. In most cases, particularly if fever is low, the child is well. In about 5�15% of young children, high fevers may trigger febrile seizures.

* Rotor syndrome is a relatively mild condition characterized by elevated levels of a substance called bilirubin in the blood (hyperbilirubinemia). Bilirubin is produced when red blood cells are broken down. It has an orange-yellow tint, and buildup of this substance can cause yellowing of the skin or whites of the eyes (jaundice). In people with Rotor syndrome, jaundice is usually evident shortly after birth or in childhood and may come and go; yellowing of the whites of the eyes (also called conjunctival icterus) is often the only symptom. There are two forms of bilirubin in the body: a toxic form called unconjugated bilirubin and a nontoxic form called conjugated bilirubin. People with Rotor syndrome have a buildup of both unconjugated and conjugated bilirubin in their blood, but the majority is conjugated.

* Rubella is a viral illness that causes a skin rash and joint pain. A rubella infection is mild for most people, but it can have catastrophic consequences for an unborn baby. If a pregnant woman contracts rubella, her baby is at risk of severe and permanent birth defects or death. Rubella is sometimes called German measles, but rubella is a different viral disease to measles. About half of rubella cases are so mild that there are no symptoms. If symptoms do occur, they usually appear between two and three weeks after infection. Some of the signs and symptoms of rubella may include: mild fever, headache, runny nose, sore eyes, skin rash, swollen lymph nodes, joint pain. Rubella is a mild illness compared to measles and most people recover within about three days. Possible complications of rubella include: arthralgia � lingering joint pain that may take a month or more to get better, otitis media � inflammation of the middle ear, encephalitis � inflammation of the brain, which can be fatal in some cases. Rubella is most commonly spread when someone ingests (swallows) or inhales the cough or sneeze droplets from an infected person. Infants with CRS shed the rubella virus in their nose and throat secretions and in their urine for months or even years. Symptoms occur usually between 14 to 17 days (and up to 21 days). People infected with rubella are infectious for approximately one week before, and for at least four days after, the onset of the rash.

* Rubinstein-Taybi syndrome is a condition characterized by short stature, moderate to severe intellectual disability, distinctive facial features, and broad thumbs and first toes. Additional features of the disorder can include eye abnormalities, heart and kidney defects, dental problems, and obesity. These signs and symptoms vary among affected individuals. People with this condition have an increased risk of developing noncancerous and cancerous tumors, including certain kinds of brain tumors. Cancer of blood-forming tissue (leukemia) also occurs more frequently in people with Rubinstein-Taybi syndrome. Rarely, Rubinstein-Taybi syndrome can involve serious complications such as a failure to gain weight and grow at the expected rate (failure to thrive) and life-threatening infections. Infants born with this severe form of the disorder usually survive only into early childhood.